<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0016-3813</journal-id>
<journal-title><![CDATA[Gaceta médica de México]]></journal-title>
<abbrev-journal-title><![CDATA[Gac. Méd. Méx]]></abbrev-journal-title>
<issn>0016-3813</issn>
<publisher>
<publisher-name><![CDATA[Academia Nacional de Medicina de México A.C.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0016-38132022000600372</article-id>
<article-id pub-id-type="doi">10.24875/gmm.22000124</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Mecanismo de células dendríticas derivadas de sangre periférica humana en la regulación de la autofagia en células de carcinoma]]></article-title>
<article-title xml:lang="en"><![CDATA[Mechanism of human peripheral blood-derived dendritic cells in the regulation of autophagy in carcinoma cells]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Liu]]></surname>
<given-names><![CDATA[Yiming]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Song]]></surname>
<given-names><![CDATA[Mengqiang]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Yuan]]></surname>
<given-names><![CDATA[Hao]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Xiao]]></surname>
<given-names><![CDATA[Xianpei]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Shu]]></surname>
<given-names><![CDATA[Jinghao]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Pan]]></surname>
<given-names><![CDATA[Wanlong]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad de Medicina del Norte de Sichuan Facultad de Medicina Básica ]]></institution>
<addr-line><![CDATA[Nanchong ]]></addr-line>
<country>China</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2022</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2022</year>
</pub-date>
<volume>158</volume>
<numero>6</numero>
<fpage>372</fpage>
<lpage>379</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S0016-38132022000600372&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S0016-38132022000600372&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S0016-38132022000600372&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen  Introducción: Las células dendríticas (CD) están involucradas en el reconocimiento, respuesta y modulación inmunológicos relacionados con la aparición del cáncer.  Objetivo: Explorar el mecanismo de las CD en la inhibición de la autofagia de las células del hepatoma.  Métodos: Células mononucleares de sangre periférica humana se aislaron mediante centrifugación en gradiente de densidad de Ficoll y se indujeron en CD, las cuales fueron cocultivadas con células HepG2 por ensayo de migración Transwell. La actividad de las células HepG2 se determinó mediante ensayo CCK8. La expresión del índice de autofagia LC3 se midió con análisis de transferencia Western y la expresión y secreción de citocinas mediante qRT-PCR y ELISA.  Resultados: En el sistema de cocultivo, las CD redujeron la viabilidad de HepG2; la expresión de IL-2, IL-12, IL-10 e IFN-&#947; en CD también se inhibió significativamente, si bien IL-2 e IFN-&#947; aún se expresaron 0.6 y 0.53 más que en el grupo de control.  Conclusión: Las CD pueden regular la autofagia de las células del carcinoma hepatocelular. El mecanismo puede estar relacionado con la síntesis y liberación de citocinas como IL-2, IL-12 e IFN-&#947; por parte de las CD.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract  Introduction: Dendritic cells (DC) are involved in immune recognition, response and immunomodulation mechanisms related to the onset of cancer.  Objective: To explore DCs mechanism in the inhibition of autophagy in hepatoma cells.  Methods: Human peripheral blood mononuclear cells were isolated by Ficoll density gradient centrifugation and induced into DCs, which were co-cultured with HepG2 cells by Transwell migration assay. HepG2 cell activity was determined using the CCK8 assay. LC3 autophagy index expression was measured with Western blot analysis, and the expression and secretion of cytokines, with qRT-PCR and ELISA.  Results: In the co-culture system, DCs were able to reduce HepG2 cells viability; IL-2, IL-12, IL-10 and IFN-&#947; expression in DCs was also significantly inhibited, although IL-2 and IFN-&#947; were still expressed 0.6 and 0.53 more than in the control group.  Conclusion: DCs can regulate autophagy in hepatocellular carcinoma cells. The mechanism may be related to the synthesis and release of cytokines such as IL-2, IL-12 and IFN-&#947; by DCs.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Autofagia]]></kwd>
<kwd lng="es"><![CDATA[Células de carcinoma]]></kwd>
<kwd lng="es"><![CDATA[Células dendríticas]]></kwd>
<kwd lng="es"><![CDATA[HepG2]]></kwd>
<kwd lng="en"><![CDATA[Autophagy]]></kwd>
<kwd lng="en"><![CDATA[Carcinoma cells]]></kwd>
<kwd lng="en"><![CDATA[Dendritic cells]]></kwd>
<kwd lng="en"><![CDATA[HepG2]]></kwd>
</kwd-group>
</article-meta>
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