<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2565-005X</journal-id>
<journal-title><![CDATA[Gaceta mexicana de oncología]]></journal-title>
<abbrev-journal-title><![CDATA[Gac. mex. oncol.]]></abbrev-journal-title>
<issn>2565-005X</issn>
<publisher>
<publisher-name><![CDATA[Sociedad Mexicana de Oncología A.C.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2565-005X2024000200084</article-id>
<article-id pub-id-type="doi">10.24875/j.gamo.24000105</article-id>
<title-group>
<article-title xml:lang="en"><![CDATA[Gastric cancer and the omics]]></article-title>
<article-title xml:lang="es"><![CDATA[Cáncer gástrico y las ómicas]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Dwivedi]]></surname>
<given-names><![CDATA[Sanyog]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Montaño-Estrada]]></surname>
<given-names><![CDATA[Luis F.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Rendón-Huerta]]></surname>
<given-names><![CDATA[Erika P.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Immunobiology Laboratory, UNAM Department of Cellular and Tissue Biology School of Medicine]]></institution>
<addr-line><![CDATA[Mexico City ]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>06</month>
<year>2024</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>06</month>
<year>2024</year>
</pub-date>
<volume>23</volume>
<numero>2</numero>
<fpage>84</fpage>
<lpage>91</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S2565-005X2024000200084&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S2565-005X2024000200084&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S2565-005X2024000200084&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Gastric cancer is a fatal process whose risk factors include infection by Helicobacter pylori, pernicious anemia, nitroso compounds, alcohol abuse, cigarette smoking, and male gender. Endoscopic surveillance has defined the histological progression of premalignant to malignant lesions. Nevertheless, gastric tumors exhibit distinct histologic variations, clinical behaviors, and treatment responses. This &#8220;intra and interpatient heterogeneity&#8221; has obliged us to search for key principles governing gastric cancer evolution. Advanced bioinformatics programs, DNA microarray technology, and functional genomics have helped to integrate the structure, function, and dynamics of biological molecules expressed or secreted by cancer epithelial cells that have modified the classification of gastric cancer.]]></p></abstract>
<abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen El cáncer gástrico es un proceso fatal cuyos factores de riesgo incluyen la infección por Helicobacter pylori, la anemia perniciosa, la ingesta de compuestos nitrosos, el abuso de alcohol, el tabaquismo y el sexo masculino. El seguimiento endoscópico ha definido la progresión histológica de lesiones premalignas a lesiones malignas. Sin embargo, los tumores gástricos exhiben diferentes y muy variadas variaciones histológicas, comportamientos clínicos y respuestas a tratamiento. Esta gran heterogeneidad intrapaciente e interpaciente ha obligado a la búsqueda de principios reguladores que gobiernen la evolución del cáncer gástrico. Los programas avanzados de bioinformática, la tecnología de microarreglos de ADN y la genómica funcional han ayudado a integrar la estructura, función y dinámica de moléculas biológicas expresadas o secretadas por las células epiteliales cancerosas que han modificado la clasificación del cáncer gástrico.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Gastric cancer]]></kwd>
<kwd lng="en"><![CDATA[Omics classification]]></kwd>
<kwd lng="en"><![CDATA[GC diversity]]></kwd>
<kwd lng="en"><![CDATA[Bioinformatics]]></kwd>
<kwd lng="es"><![CDATA[Cáncer gástrico]]></kwd>
<kwd lng="es"><![CDATA[Clasificación ómicas]]></kwd>
<kwd lng="es"><![CDATA[Diversidad del cáncer gástrico]]></kwd>
<kwd lng="es"><![CDATA[Bioinformática]]></kwd>
</kwd-group>
</article-meta>
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