<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1665-5044</journal-id>
<journal-title><![CDATA[Revista mexicana de neurociencia]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. mex. neurocienc.]]></abbrev-journal-title>
<issn>1665-5044</issn>
<publisher>
<publisher-name><![CDATA[Academia Mexicana de Neurología A.C.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1665-50442025000600006</article-id>
<article-id pub-id-type="doi">10.24875/rmn.25000003</article-id>
<title-group>
<article-title xml:lang="en"><![CDATA[Alterations of the GABAergic system in binge eating disorder: a systematic review]]></article-title>
<article-title xml:lang="es"><![CDATA[Alteraciones del sistema GABAérgico en el trastorno por atracón: una revisión sistemática]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Hage-Barritta]]></surname>
<given-names><![CDATA[Natalia M. El]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Serafini]]></surname>
<given-names><![CDATA[Matías]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Cuenya]]></surname>
<given-names><![CDATA[Lucas]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Pedrón]]></surname>
<given-names><![CDATA[Valeria T.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Abierta Interamericana Centro de Altos Estudios en Ciencias Humanas y de la Salud ]]></institution>
<addr-line><![CDATA[Buenos Aires ]]></addr-line>
<country>Argentina</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Grupo interdisciplinario de investigación en aprendizaje y conductas de ingesta durante el desarrollo  ]]></institution>
<addr-line><![CDATA[Santiago ]]></addr-line>
<country>Chile</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Instituto de Biología Molecular y Neurociencias Laboratorio de Neurofisiología de la Memoria ]]></institution>
<addr-line><![CDATA[Santiago ]]></addr-line>
<country>Chile</country>
</aff>
<aff id="Af4">
<institution><![CDATA[,Universidad de Chile Facultad de Ciencias Sociales Departamento de Psicología]]></institution>
<addr-line><![CDATA[Santiago ]]></addr-line>
<country>Chile</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2025</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2025</year>
</pub-date>
<volume>26</volume>
<numero>6</numero>
<fpage>204</fpage>
<lpage>215</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S1665-50442025000600006&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S1665-50442025000600006&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S1665-50442025000600006&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Binge eating disorder (BED) is characterized by recurrent episodes of excessive intake within a limited time period, accompanied by loss of control and distress. These behaviors may overstimulate and desensitize the dopaminergic system, leading to the search for palatable foods to reactivate a hypofunctional reward circuit. Dopamine release activates GABAergic pathways, which exert inhibitory control by reducing dopamine release, suggesting that this system may be altered in the context of BED. The objective of this systematic review was to gather research conducted between 2013 and 2023 on GABAergic functioning in eating behavior and binge episodes, focusing exclusively on rodent models (rats and mice). The review followed PRISMA 2020 guidelines, using ScienceDirect and PubMed as data sources. The initial search yielded 488 results. After screening, 23 articles were selected, and five met the inclusion criteria. Of these, 60% (n = 3) were conducted in mice and 40% (n = 2) in rats. In 80% of cases (n = 4), only male animals were used, while 20% (n = 1) included both sexes. Likewise, 80% (n = 4) used adult animals, and 20% (n = 1) did not report age. Contradictory results emerged regarding GABA involvement in feeding behavior: some studies showed increased intake associated with both lower and higher GABAergic activation, while others reported the opposite pattern. These inconsistencies may reflect interactions between distinct neuroanatomical regions and procedural differences. Most studies focused on general food intake modulation rather than binge-specific behaviors, limiting the direct applicability of findings to BED.]]></p></abstract>
<abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen El Trastorno por Atracón se caracteriza por episodios recurrentes de ingesta excesiva en un período de tiempo limitado, acompañados por pérdida de control y malestar significativo. Estos episodios pueden generar sobreestimulación y posterior desensibilización del sistema dopaminérgico, favoreciendo la búsqueda de alimentos palatables para reactivar un sistema de recompensa hipofuncional. La liberación de dopamina activa vías GABAérgicas inhibidoras que reducen su propia liberación, lo que sugiere posibles alteraciones en este sistema. El objetivo de esta revisión sistemática fue recopilar investigaciones realizadas entre 2013 y 2023 sobre el funcionamiento GABAérgico en la conducta alimentaria y en episodios de atracón. Se consideraron únicamente estudios en modelos animales (ratas o ratones). Se siguieron las directrices de PRISMA 2020, utilizando ScienceDirect y PubMed. La búsqueda inicial arrojó 488 resultados; tras la selección, 23 artículos fueron evaluados y 5 cumplieron con los criterios de inclusión. De ellos, el 60% (n = 3) se realizó en ratones y el 40% (n = 2) en ratas. En el 80% (n = 4) se utilizaron machos y en el 20% (n = 1) ambos sexos. El 80% (n = 4) empleó animales adultos y uno no informó la edad. Se identificaron resultados contradictorios: algunos estudios mostraron mayor ingesta con menor o mayor activación GABAérgica, mientras que otros evidenciaron el patrón inverso. Esta ambigüedad podría deberse a diferencias neuroanatómicas y metodológicas. La mayoría abordó la modulación general de la ingesta, lo que limita la extrapolación directa de los hallazgos al fenotipo conductual del trastorno por atracón.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Binge eating disorder]]></kwd>
<kwd lng="en"><![CDATA[GABAergic system]]></kwd>
<kwd lng="en"><![CDATA[Dopaminergic system]]></kwd>
<kwd lng="en"><![CDATA[Neurobiological mechanisms]]></kwd>
<kwd lng="en"><![CDATA[Eating behavior]]></kwd>
<kwd lng="es"><![CDATA[Trastorno por atracón]]></kwd>
<kwd lng="es"><![CDATA[Sistema GABAérgico]]></kwd>
<kwd lng="es"><![CDATA[Sistema dopaminérgico]]></kwd>
<kwd lng="es"><![CDATA[Mecanismos neurobiológicos]]></kwd>
<kwd lng="es"><![CDATA[Conducta alimentaria]]></kwd>
</kwd-group>
</article-meta>
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