<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1665-5044</journal-id>
<journal-title><![CDATA[Revista mexicana de neurociencia]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. mex. neurocienc.]]></abbrev-journal-title>
<issn>1665-5044</issn>
<publisher>
<publisher-name><![CDATA[Academia Mexicana de Neurología A.C.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1665-50442019000100021</article-id>
<article-id pub-id-type="doi">10.24875/rmn.m19000023</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Neurobiología del trastorno de estrés postraumático]]></article-title>
<article-title xml:lang="en"><![CDATA[Neurobiology review of post-traumatic stress disorder]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Zegarra-Valdivia]]></surname>
<given-names><![CDATA[Jonathan A.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Chino-Vilca]]></surname>
<given-names><![CDATA[Brenda N.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Nacional de San Agustín de Arequipa  ]]></institution>
<addr-line><![CDATA[Arequipa ]]></addr-line>
<country>Perú</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad Católica San Pablo  ]]></institution>
<addr-line><![CDATA[Arequipa ]]></addr-line>
<country>Perú</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>02</month>
<year>2019</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>02</month>
<year>2019</year>
</pub-date>
<volume>20</volume>
<numero>1</numero>
<fpage>21</fpage>
<lpage>28</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S1665-50442019000100021&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S1665-50442019000100021&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S1665-50442019000100021&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen A lo largo de la vida, las personas suelen estar expuestas a situaciones traumáticas, lo que en muchas ocasiones provoca la aparición del trastorno de estrés postraumático (TEPT), cuya prevalencia alcanza del 5 al 10% a nivel mundial. Sin embargo, pese a la gran cantidad de investigaciones que abordan el trastorno, la evidencia científica respecto a los procesos neurobiológicos aún no es concluyente. La presente revisión busca profundizar en el conocimiento de las características neurobiológicas más relevantes de los pacientes con TEPT. Se realiza una descripción de las áreas cerebrales involucradas y los efectos del TEPT en su funcionamiento, incluyendo un análisis del compromiso activo del eje hipotálamico-hipofisiario-adrenal, la hiperactivación y excesiva respuesta adrenérgica en el sistema nervioso autónomo, además de una breve descripción de los modelos animales más usados en las investigaciones. Se debe de tener en cuenta que la mayoría de los trastornos neuropsiquiátricos no comportan la alteración específica de una red neural, área o sistema, sino que en su conjunto presentan una alteración de distintos sistemas simultáneamente, que se solapan, y a su vez se hacen evidentes con endofenotipos neurocognitivos relativamente específicos, que apoyados en la investigación básica y la utilización con modelos animales proporcionaría los conocimientos de la alteración celular y molecular relacionada, aspectos significativos en la evaluación, tratamiento y seguimiento de los pacientes.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Throughout life, people are often exposed to traumatic situations, causing the onset of post-traumatic stress disorder (PTSD), whose prevalence reaches 5-10% worldwide. However, despite the large amount of research that addresses the disorder, the scientific evidence regarding neurobiological processes is not yet conclusive. The present review seeks to deep in the knowledge of the most relevant neurobiological characteristics of patients with PTSD. A description is made of the brain areas involved and the effects of PTSD on its functioning, including an analysis of the active commitment of the hypothalamic-pituitary-adrenal axis, hyperactivation and excessive adrenergic response in the autonomic nervous system, as well as a brief description of the animal models most used in research. It must be taken into account that the majority of neuropsychiatric disorders do not involve the specific alteration of a neural network, area or system, but as a whole they present an alteration of different systems simultaneously, which overlap and in turn become evident with relatively specific neurocognitive endophenotypes that, supported by basic research and use with animal models, would provide knowledge of the related cellular and molecular alteration, significant aspects in the evaluation, treatment, and follow-up of patients.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Estrés]]></kwd>
<kwd lng="es"><![CDATA[Trastorno de estrés postraumático]]></kwd>
<kwd lng="es"><![CDATA[Neurobiología]]></kwd>
<kwd lng="es"><![CDATA[Modelos animales]]></kwd>
<kwd lng="es"><![CDATA[Revisión]]></kwd>
<kwd lng="en"><![CDATA[Animal models]]></kwd>
<kwd lng="en"><![CDATA[Post-traumatic stress disorder]]></kwd>
<kwd lng="en"><![CDATA[Neurobiology]]></kwd>
<kwd lng="en"><![CDATA[Review]]></kwd>
<kwd lng="en"><![CDATA[Stress]]></kwd>
</kwd-group>
</article-meta>
</front><back>
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