<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1405-9940</journal-id>
<journal-title><![CDATA[Archivos de cardiología de México]]></journal-title>
<abbrev-journal-title><![CDATA[Arch. Cardiol. Méx.]]></abbrev-journal-title>
<issn>1405-9940</issn>
<publisher>
<publisher-name><![CDATA[Instituto Nacional de Cardiología Ignacio Chávez]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1405-99402025000100034</article-id>
<article-id pub-id-type="doi">10.24875/acm.24000065</article-id>
<title-group>
<article-title xml:lang="en"><![CDATA[Association of the rs12617656 C/T genetic variant of the DPP4 gene with in-stent restenosis in Mexican population: a cohort study]]></article-title>
<article-title xml:lang="es"><![CDATA[Asociación de la variante genética rs12617656 C/T del gen DPP4 con restenosis intra-stent en población mexicana: estudio de corte]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Vargas-Alarcón]]></surname>
<given-names><![CDATA[Gilberto]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Posadas-Sanchez]]></surname>
<given-names><![CDATA[Rosalinda]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Martínez-Ríos]]></surname>
<given-names><![CDATA[Marco A.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Delgadillo-Rodriguez]]></surname>
<given-names><![CDATA[Hilda]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[López-Olmos]]></surname>
<given-names><![CDATA[Victoria]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Fragoso]]></surname>
<given-names><![CDATA[José M.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Instituto Nacional de Cardiología Ignacio Chávez Departamento de Biología Molecular ]]></institution>
<addr-line><![CDATA[ Ciudad de México]]></addr-line>
<country>México</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Instituto Nacional de Cardiología Ignacio Chávez Departamento de Endocrinología ]]></institution>
<addr-line><![CDATA[ Ciudad de México]]></addr-line>
<country>México</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Instituto Nacional de Cardiología Ignacio Chávez Departamento de Hemodinámica ]]></institution>
<addr-line><![CDATA[ Ciudad de México]]></addr-line>
<country>México</country>
</aff>
<aff id="Af4">
<institution><![CDATA[,Instituto Nacional de Cardiología Ignacio Chávez Departamento de Consulta Externa ]]></institution>
<addr-line><![CDATA[ Ciudad de México]]></addr-line>
<country>México</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>03</month>
<year>2025</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>03</month>
<year>2025</year>
</pub-date>
<volume>95</volume>
<numero>1</numero>
<fpage>34</fpage>
<lpage>41</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S1405-99402025000100034&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S1405-99402025000100034&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S1405-99402025000100034&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract  Objective: We evaluated whether five (rs12617336, rs12617656, rs1558957, rs3788979, and rs17574) single nucleotide polymorphisms located in the DPP4 gene that have not been sufficiently explored, are candidates to be risk markers of in-stent restenosis.  Methods: The genotypes were determined in 190 patients (60 patients with restenosis and 130 without restenosis) using 5&#8217;exonuclease TaqMan assays in accordance with the manufacturer&#8217;s instructions (Applied Biosystems, Foster City, USA).  Results: The results showed that the CC genotype of the rs12617656 C/T SNP was associated with the risk of development restenosis after coronary stent under the co-dominant, and recessive models (odds ratios [OR] = 3.32, p = 0.0009, and OR = 4.96, p = 0.0008, respectively). In addition, our data showed that the genetic distribution of the rs12617336, rs1558957, rs3788979, and rs17574 SNPs were similar between patients with and without restenosis after coronary stenting. On the other hand, the haplotype analysis showed one haplotype (CTC) associated with restenosis susceptibility (p = 0.006).  Conclusion: Our study demonstrates that the rs12617656 genetic variant of the DPP4 gene is associated with the risk of developing in-stent restenosis in our population.]]></p></abstract>
<abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen  Objetivo: En este estudio nosotros evaluamos si cinco polimorfismos de un solo nucleótido localizados en el gen DPP4 (rs12617336, rs12617656, rs1558957, rs3788979, y rs17574), que no han sido lo suficientemente explorados, podrían ser marcadores de riesgo para la restenosis intra-stent.  Métodos: Los genotipos se determinaron en 190 pacientes (60 con restenosis y 130 sin restenosis) usando ensayos TaqMan 5&#8217; exonucleasa de acuerdo con las instrucciones del fabricante (Applied Biosystems, foster City, USA).  Resultados: Los resultados mostraron que el genotipo CC del polimorfismo rs12617656 C/T fue asociada con riesgo de desarrollar restenosis intra-stent, bajo los modelos de herencia co-dominant y recesivo (OR = 3.32, p = 0.0009, and OR = 4.96, p = 0.0008, respectivamente). Asimismo, nuestros datos mostraron que la distribución de los sitios polimórficos rs12617336, rs1558957, rs3788979, y rs17574 fue similar en pacientes con y sin restenosis después del implante del stent coronario. Por otro lado, el análisis de haplotipos mostro que el haplotipo (CTC) es asociado con la susceptibilidad a desarrollar restenosis intra-stent. (p = 0.006).  Conclusión: Nuestros datos demostraron que el polimorfismo rs12617656 C/T es asociado con el riesgo de desarrollar restenosis intra-stent en nuestra población.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Association]]></kwd>
<kwd lng="en"><![CDATA[Single nucleotide polymorphisms]]></kwd>
<kwd lng="en"><![CDATA[In-stent restenosis]]></kwd>
<kwd lng="es"><![CDATA[Asociación]]></kwd>
<kwd lng="es"><![CDATA[Polimorfismos de un solo nucleótido]]></kwd>
<kwd lng="es"><![CDATA[Restenosis intra-stent]]></kwd>
</kwd-group>
</article-meta>
</front><back>
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<given-names><![CDATA[J]]></given-names>
</name>
<name>
<surname><![CDATA[Tanaka]]></surname>
<given-names><![CDATA[T]]></given-names>
</name>
<name>
<surname><![CDATA[Nojima]]></surname>
<given-names><![CDATA[Y]]></given-names>
</name>
<name>
<surname><![CDATA[Schlossman]]></surname>
<given-names><![CDATA[SF]]></given-names>
</name>
<name>
<surname><![CDATA[Morimoto]]></surname>
<given-names><![CDATA[C]]></given-names>
</name>
</person-group>
<article-title xml:lang=""><![CDATA[Direct association of adenosine deaminase with a T cell activation antigen, CD26]]></article-title>
<source><![CDATA[Science]]></source>
<year>1993</year>
<volume>261</volume>
<page-range>466-9</page-range></nlm-citation>
</ref>
</ref-list>
</back>
</article>
