<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0484-7903</journal-id>
<journal-title><![CDATA[Revista mexicana de anestesiología]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. mex. anestesiol.]]></abbrev-journal-title>
<issn>0484-7903</issn>
<publisher>
<publisher-name><![CDATA[Colegio Mexicano de Anestesiología A.C.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0484-79032025000400225</article-id>
<article-id pub-id-type="doi">10.35366/121397</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Cambios ultraestructurales en las mitocondrias de pacientes diabéticos sometidos a cirugía cardíaca con circulación extracorpórea]]></article-title>
<article-title xml:lang="en"><![CDATA[Ultrastructural changes in the mitochondria of diabetic patients undergoing cardiac surgery with extracorporeal circulation]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Luna-Ortiz]]></surname>
<given-names><![CDATA[Pastor]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Soria-Castro]]></surname>
<given-names><![CDATA[Elizabeth]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Soule-Egea]]></surname>
<given-names><![CDATA[Mauricio]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Gómez-Sánchez]]></surname>
<given-names><![CDATA[Mario]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Martínez-Rosas]]></surname>
<given-names><![CDATA[Martín]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Torrico-Lavayen]]></surname>
<given-names><![CDATA[Rocío]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Instituto Nacional de Cardiología Ignacio Chávez Departamento de Farmacología ]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>México</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Instituto Nacional de Cardiología Ignacio Chávez Departamento de Patología ]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>México</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Instituto Nacional de Cardiología Ignacio Chávez Departamento de Cirugía ]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>México</country>
</aff>
<aff id="Af4">
<institution><![CDATA[,Instituto Nacional de Cardiología Ignacio Chávez Departamento de Fisiología ]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>México</country>
</aff>
<aff id="Af5">
<institution><![CDATA[,Instituto Nacional de Cardiología Ignacio Chávez Departamento de Patología ]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>México</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2025</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2025</year>
</pub-date>
<volume>48</volume>
<numero>4</numero>
<fpage>225</fpage>
<lpage>228</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S0484-79032025000400225&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S0484-79032025000400225&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S0484-79032025000400225&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen: Los pacientes con diabetes mellitus tipo 2 (DM2) presentan mayor incidencia de enfermedad cardiovascular, en particular insuficiencia cardíaca (IC), como parte de la miocardiopatía diabética (MCD). Actualmente se reconoce que los trastornos en el metabolismo energético cardíaco tienen un papel fundamental en el desarrollo de la IC de la MCD. La disfunción mitocondrial de los cardiomiocitos podría explicar la patogénesis de la IC presente en esta enfermedad. Existen diversos estudios a nivel experimental de las alteraciones en la estructura y función mitocondrial en diversos tejidos de modelos animales que explican las alteraciones funcionales miocárdicas, sin embargo, existen pocos estudios en sujetos humanos. El objetivo de este trabajo fue estudiar los cambios ultraestructurales en las mitocondrias de los apéndices de la aurícula derecha del corazón de pacientes diabéticos operados de cirugía cardíaca con circulación extracorpórea. Los resultados muestran una disminución en el número de mitocondrias, así como su morfología, tamaño y distribución en las muestras de los pacientes con DM2 comparado con no diabéticos. Aunque este estudio demuestra claramente los cambios ultraestructurales mitocondriales, se requieren estudios adicionales que demuestren la disfunción mitocondrial en el corazón diabético.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract: Patients with type 2 diabetes mellitus (T2DM) have a higher incidence of cardiovascular disease, particularly heart failure (HF), as part of diabetic cardiomyopathy (DCM). It is now recognized that disorders in cardiac energy metabolism play a fundamental role in the development of HF from DCM. Mitochondrial dysfunction of cardiomyocytes could explain the pathogenesis of HF present in these patients. There are several experimental studies on alterations in mitochondrial structure and function in various tissues of animal models that explain myocardial functional alterations. However, there are few studies on human subjects. The objective of this work was to study the ultrastructural changes in the mitochondria of the appendages of the right atrium of the heart of diabetic patients undergoing cardiac surgery with extracorporeal circulation. The results show a decrease in the number of mitochondria, as well as their morphology, size, and distribution in samples from patients with T2DM compared to non-diabetics. Although this study demonstrates mitochondrial ultrastructural changes, functional studies are required to demonstrate mitochondrial dysfunction in the diabetic heart.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[mitocondrias cardíacas]]></kwd>
<kwd lng="es"><![CDATA[cardiomiopatía diabética]]></kwd>
<kwd lng="es"><![CDATA[insuficiencia cardíaca]]></kwd>
<kwd lng="es"><![CDATA[disfunción mitocondrial]]></kwd>
<kwd lng="en"><![CDATA[cardiac mitochondria]]></kwd>
<kwd lng="en"><![CDATA[diabetic cardiomyopathy]]></kwd>
<kwd lng="en"><![CDATA[heart failure]]></kwd>
<kwd lng="en"><![CDATA[mitochondrial dysfunction]]></kwd>
</kwd-group>
</article-meta>
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