<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2444-054X</journal-id>
<journal-title><![CDATA[Cirugía y cirujanos]]></journal-title>
<abbrev-journal-title><![CDATA[Cir. cir.]]></abbrev-journal-title>
<issn>2444-054X</issn>
<publisher>
<publisher-name><![CDATA[Academia Mexicana de Cirugía A.C.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2444-054X2025000100006</article-id>
<article-id pub-id-type="doi">10.24875/ciru.23000304</article-id>
<title-group>
<article-title xml:lang="en"><![CDATA[Exenatide improves cisplatin induced ovarian damage through NLRP3, Nrf-2, and TLR4 pathways]]></article-title>
<article-title xml:lang="es"><![CDATA[La exenatida mejora el daño ovárico inducido por cisplatino a través de las vías NLRP3, Nrf-2 y TLR4]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Do&#287;an]]></surname>
<given-names><![CDATA[Gül O.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Erba&#351;]]></surname>
<given-names><![CDATA[Oytun]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,&#350;i&#351;li Hamidiye Eftal Hospital Department of Gynecology and Obstetrics ]]></institution>
<addr-line><![CDATA[Istanbul ]]></addr-line>
<country>Turkey</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Demiro&#287;lu Bilim University Medical School Department of Physiology ]]></institution>
<addr-line><![CDATA[Istanbul ]]></addr-line>
<country>Turkey</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>02</month>
<year>2025</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>02</month>
<year>2025</year>
</pub-date>
<volume>93</volume>
<numero>1</numero>
<fpage>35</fpage>
<lpage>40</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S2444-054X2025000100006&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S2444-054X2025000100006&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S2444-054X2025000100006&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract  Objective: Cisplatin (CP) toxicity causes ovarian damage by oxidative stress, inflammation and fibrosis. The aim of the present study is to investigate the possible beneficial effects of exenatide on the experimental ovarian damage model produced by CP.  Method: For 14 rats, CP was administered by intraperitoneally (i.p) twice a week for 5 weeks. No drug was administered to the remainder of rats (n = 7) (Group 0). The rats taken CP were divided into two groups. Group 1 rats (n = 7) were given 1 mL/kg/day saline i.p., and Group 2 rats (n = 7) was given with 20 &#956;g/kg/day exenatide.  Results: The number of primordial, primary, secondary, and tertiary follicle was significantly lower in Group1 compared with Group 0 and Group 2. The ovarian fibrosis percent was significantly higher in Group 1 than Group 0 and 2. The plasma anti-Mullerian hormone value was lower in Group1compared with Group 0 and 2. Over Nuclear factor-erythroid factor 2-related factor 2 level, Over Toll-like receptor 4 level and over nucleotide-binding domain leucine-rich repeat and pyrin domain containing receptor 3 were higher in Group 1 compared with Group 0 and 2.  Conclusion: exenatide has possible beneficial effect on ovarian damage induced by CP by anti-inflammatory actions and can be a promising candidate for ovarian damage caused by CP.]]></p></abstract>
<abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen  Objetivo: La toxicidad del cisplatino causa daño ovárico por estrés oxidativo, inflamación y fibrosis. El objetivo del presente estudio fue investigar los posibles efectos beneficiosos de la exenatida en el modelo experimental de daño ovárico producido por el cisplatino.  Método: A 14 ratas se les administró cisplatino por vía intraperitoneal (i.p.) dos veces por semana durante 5 semanas. No se administró fármaco al resto de la ratas (n = 7, grupo 0). Las ratas que recibieron cisplatino se dividieron en dos grupos. Las ratas del grupo 1 (n = 7) recibieron 1 ml/kg/día de solución salina i.p. y las ratas del grupo 2 (n = 7) recibieron 20 &#956;g/kg/día de exenatida.  Resultados: El número de folículos primordiales, primarios, secundarios y terciarios, fue significativamente menor en el grupo 1 en comparación con los grupos 0 y 2. El nivel superior a NrF2, el nivel superior a TLR-4 y el nivel superior a NLRP-3 fueron más altos en el grupo 1 en comparación con los grupos 0 y 2.  Conclusión: La exenatida tiene un posible efecto beneficioso sobre el daño ovárico inducido por el cisplatino por acciones antiinflamatorias, y puede ser un agente candidato prometedor para el daño ovárico causado por el cisplatino.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Cisplatin]]></kwd>
<kwd lng="en"><![CDATA[Exenatide]]></kwd>
<kwd lng="en"><![CDATA[Ovarian]]></kwd>
<kwd lng="en"><![CDATA[Toxicity]]></kwd>
<kwd lng="es"><![CDATA[Cisplatino]]></kwd>
<kwd lng="es"><![CDATA[Exenatida]]></kwd>
<kwd lng="es"><![CDATA[Ovario]]></kwd>
<kwd lng="es"><![CDATA[Toxicidad]]></kwd>
</kwd-group>
</article-meta>
</front><back>
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