<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1405-888X</journal-id>
<journal-title><![CDATA[TIP. Revista especializada en ciencias químico-biológicas]]></journal-title>
<abbrev-journal-title><![CDATA[TIP]]></abbrev-journal-title>
<issn>1405-888X</issn>
<publisher>
<publisher-name><![CDATA[Universidad Nacional Autónoma de México, Facultad de Estudios Superiores Zaragoza]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1405-888X2021000100103</article-id>
<article-id pub-id-type="doi">10.22201/fesz.23958723e.2021.302</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Tratamiento a largo plazo con campos electromagnéticos en un modelo animal de la enfermedad de Parkinson]]></article-title>
<article-title xml:lang="en"><![CDATA[Long-term treatment with electromagnetic fields in an animal model of Parkinson´s disease]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Medina-Salazar]]></surname>
<given-names><![CDATA[Idalia]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Moreno-Fitz]]></surname>
<given-names><![CDATA[Jacqueline]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Jiménez]]></surname>
<given-names><![CDATA[Grecia]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Morales]]></surname>
<given-names><![CDATA[Noé]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Pizarro]]></surname>
<given-names><![CDATA[Monserrat]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Elías-Viñas]]></surname>
<given-names><![CDATA[David]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Verdugo-Díaz]]></surname>
<given-names><![CDATA[Leticia]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Nacional Autónoma de México Facultad de Medicina Departamento de Fisiología]]></institution>
<addr-line><![CDATA[Ciudad de México ]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Instituto Politécnico Nacional Centro de Investigación y de Estudios Avanzados ]]></institution>
<addr-line><![CDATA[Ciudad de México ]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>00</month>
<year>2021</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>00</month>
<year>2021</year>
</pub-date>
<volume>24</volume>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S1405-888X2021000100103&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S1405-888X2021000100103&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S1405-888X2021000100103&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen La enfermedad de Parkinson (EP) es un padecimiento neurodegenerativo que afecta las neuronas de la sustancia negra pars compacta (SNc), provocando alteraciones motoras y no motoras. En la actualidad para el tratamiento de pacientes con EP se aplica la Estimulación con Campos Electromagnéticos (CEM), observándose cierta mejoría clínica. Sin embargo, su mecanismo terapéutico subyacente sigue sin ser dilucidado. Los estudios en modelos animales de la EP se han realizado por periodos cortos de estimulación con CEM. Por lo tanto, el objetivo del presente estudio fue investigar los cambios en el comportamiento motor de ratas Wistar con hemiparkinson, tratadas con CEM de 60 Hz, 2 horas diariamente durante 6 meses. Mensualmente se realizaron evaluaciones de conducta: rotación inducida por apomorfina, campo abierto, laberinto elevado en cruz y barra de equilibrio. Al finalizar el tratamiento se contó el número de neuronas dopaminérgicas en la SNc y se estimó su grado de pérdida. Los animales lesionados que recibieron CEM tuvieron una ganancia de peso similar a los animales control. El tratamiento redujo la conducta de giro y mejoró el equilibrio, sin cambios significativos en la sobrevida de las neuronas dopaminérgicas de la SNc. Estos resultados apoyan el uso de los CEM de 60 Hz como una terapia complementaria que puedan ayudar a mejorar los síntomas motores de la enfermedad.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Parkinson&#8217;s disease (PD) is a neurodegenerative disease that causes motor and non-motor alterations induced by the loss of neurons of substance nigra pars compacta (SNc) In clinical therapy, the use of pharmacological treatments to improve symptoms is commonly used. Recently, non-pharmacological techniques are being developed. Such is the case for Electromagnetic Field Stimulation (EMF). Although this treatment has already been used in PD patients, its underlying therapeutic mechanism remains unclear. Studies in animal models of PD have been performed but only for short periods of stimulation with EMF. This study aimed to investigate motor behavior in hemiparkinsonian Wistar rats treated with EMF of 60 Hz (2.4 mT) 2 hours daily for six months. Behavioral evaluations such as apomorphine-induced rotation, open field test, elevated cross maze, and balance beam were performed monthly. Also, immunoreactive dopaminergic neurons were counted, and their degree of loss was estimated. The main results showed that lesioned animals treated with EMF, did not differ in weight gain compared to control animals. Furthermore, magnetic treatment reduced turning behavior and improved balance, without significant changes in the survival of SNc dopaminergic neurons. These results support the use of EMF as an alternative therapy that could help to improve the motor symptoms of PD.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[ansiedad]]></kwd>
<kwd lng="es"><![CDATA[equilibrio]]></kwd>
<kwd lng="es"><![CDATA[hemiparkinson]]></kwd>
<kwd lng="es"><![CDATA[locomoción]]></kwd>
<kwd lng="es"><![CDATA[neuronas dopaminérgicas]]></kwd>
<kwd lng="es"><![CDATA[rata]]></kwd>
<kwd lng="en"><![CDATA[anxiety]]></kwd>
<kwd lng="en"><![CDATA[dopaminergic neurons]]></kwd>
<kwd lng="en"><![CDATA[equilibrium]]></kwd>
<kwd lng="en"><![CDATA[hemiparkinson]]></kwd>
<kwd lng="en"><![CDATA[locomotion]]></kwd>
<kwd lng="en"><![CDATA[rat]]></kwd>
</kwd-group>
</article-meta>
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