<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0016-3813</journal-id>
<journal-title><![CDATA[Gaceta médica de México]]></journal-title>
<abbrev-journal-title><![CDATA[Gac. Méd. Méx]]></abbrev-journal-title>
<issn>0016-3813</issn>
<publisher>
<publisher-name><![CDATA[Academia Nacional de Medicina de México A.C.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0016-38132025000100014</article-id>
<article-id pub-id-type="doi">10.24875/gmm.m25000961</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Alta frecuencia de síndrome metabólico en niños mayas mexicanos sin obesidad: implicaciones de las variantes genéticas de PPARG, KCNJ1, HHEX, HNF4A, ACE (I/D), FTO y ABCA1]]></article-title>
<article-title xml:lang="en"><![CDATA[High frequency of metabolic syndrome in non-obese Maya children from México: Implications of PPARG, KCNJ1, HHEX, HNF4A, ACE (I/D), FTO and ABCA1 genetics variants]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Peña-Espinoza]]></surname>
<given-names><![CDATA[Barbara]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Juárez-López]]></surname>
<given-names><![CDATA[Carlos]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Ortiz-López]]></surname>
<given-names><![CDATA[Guadalupe]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Granados-Silvestre]]></surname>
<given-names><![CDATA[Ángeles]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Menjivar]]></surname>
<given-names><![CDATA[Marta]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Autónoma de México Facultad de Química Laboratorio de Genómica de la Diabetes]]></institution>
<addr-line><![CDATA[Mérida Yucatán]]></addr-line>
<country>México</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Secretaría de Salud del Estado de Campeche Indesalud ]]></institution>
<addr-line><![CDATA[Campeche ]]></addr-line>
<country>México</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Hospital Juárez de México Laboratorio de Endocrinología Molecular ]]></institution>
<addr-line><![CDATA[Ciudad de México ]]></addr-line>
<country>México</country>
</aff>
<aff id="Af4">
<institution><![CDATA[,Universidad Nacional Autónoma de México Facultad de Química Laboratorio de Diabetes]]></institution>
<addr-line><![CDATA[Ciudad de México ]]></addr-line>
<country>México</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>02</month>
<year>2025</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>02</month>
<year>2025</year>
</pub-date>
<volume>161</volume>
<numero>1</numero>
<fpage>116</fpage>
<lpage>124</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S0016-38132025000100014&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S0016-38132025000100014&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S0016-38132025000100014&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen  Antecedentes: Factores ambientales y genéticos determinan el síndrome metabólico (SMet), el cual constituye un problema de salud nacional en adultos y niños, con mayor incidencia en individuos indígenas que en mestizos.  Objetivo: Evaluar la asociación de las variantes rs1801282, rs5219, rs1111875, rs1800961, I/D, rs9939609 y rs9282541 de los genes PPARG, KCNJ11, HHEX, HNF4A, ACE, FTO y ABCA1, respectivamente, con el SMet o sus componentes en niños mayas de Yucatán sin obesidad.  Material y métodos: Se reclutaron 508 niños de nueve a 13 años. Se utilizaron modelos univariados y multivariados ajustados por sexo, edad e índice de masa corporal.  Resultados: La frecuencia de SMet en niños mayas sin obesidad de áreas rurales y urbanas fue de 35 y 39 %, respectivamente. El análisis genotipo-fenotipo en niños mayas de áreas rurales reveló que rs9282541 se asoció a glucosa alta (p = 0.011); rs9939609, a presión arterial alta (p = 0.048); rs1800961, a insulina alta y HOMA-IR (p = 0.038, p = 0.043). En niños de áreas urbanas, I/D se asoció a presión arterial alta (p = 0.022); rs1111875, a triglicéridos altos (p = 0.050) y rs1800961, a colesterol-HDL bajo (p = 0.048).  Conclusiones: Los hallazgos proporcionan evidencia sólida del papel de las variantes estudiadas para conferir susceptibilidad genética para el desarrollo del síndrome metabólico en niños mayas sin obesidad de México.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract  Background: Both environmental and genetic factors determine metabolic syndrome (MetS) and eventually result in metabolic diseases. MetS is a national health problem in adults and children, with a higher incidence in Indigenous than mestizo individuals.  Objective: Evaluate the association of PPARG/rs1801282, KCNJ11/rs5219, HHEX/rs1111875, HNF4A/rs1800961, ACE-I/D, FTO/rs9939609 and ABCA1/rs9282541 variants with MetS or its components in the Maya children from Yucatan.  Material and methods: A total of 508 Maya children of 9 to 13 years were recruited. We analyze the association of genetic variants with MetS in non-obese Maya children by univariate and multivariate models adjusted by sex, age, and BMI.  Results: Interestingly, the frequency of MetS in non-obese Maya children from rural and urban areas was 35 % and 39 %, respectively. The genotype-phenotype analysis in rural Maya children revealed that rs9282541-ABCA1 was associated with high glucose (p = 0.011); rs9939609-FTO, with high blood pressure (p = 0.048) and rs1800961-HNF4A, with high insulin and HOMA-IR (p = 0.038, p= 0.043). In urban children, I/D-ECA was associated with high blood pressure (p = 0.022); rs1111875-HHEX, with high triglycerides (p = 0.050) and rs1800961-HNF4A, with low HDL-c (p = 0.048).  Conclusions: These findings provide strong evidence of the role of the studied variants in conferring genetic susceptibility to develop MetS in non-obese Maya children from Mexico.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Genes]]></kwd>
<kwd lng="es"><![CDATA[Niños mayas]]></kwd>
<kwd lng="es"><![CDATA[Resistencia a la insulina]]></kwd>
<kwd lng="en"><![CDATA[Genes]]></kwd>
<kwd lng="en"><![CDATA[Maya children]]></kwd>
<kwd lng="en"><![CDATA[Insulin resistance]]></kwd>
</kwd-group>
</article-meta>
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