<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2696-130X</journal-id>
<journal-title><![CDATA[Revista mexicana de angiología]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. mex. angiol.]]></abbrev-journal-title>
<issn>2696-130X</issn>
<publisher>
<publisher-name><![CDATA[Sociedad Mexicana de Angiología, Cirugía Vascular y Endovascular A.C.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2696-130X2022000100026</article-id>
<article-id pub-id-type="doi">10.24875/rma.22000001</article-id>
<title-group>
<article-title xml:lang="en"><![CDATA[Expression of miR-145, miR-146, and miR-155 in an experimental model of arteriovenous fistula]]></article-title>
<article-title xml:lang="es"><![CDATA[Expresión de miR-145, miR-146 y miR-155 en un modelo experimental de fístula arteriovenosa]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Laparra-Escareño]]></surname>
<given-names><![CDATA[Hugo]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Ortega-Gómez]]></surname>
<given-names><![CDATA[Alett]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Manzo-Merino]]></surname>
<given-names><![CDATA[Joaquín]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Zentella-Dehesa]]></surname>
<given-names><![CDATA[Alejandro]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Bobadilla-Rosado]]></surname>
<given-names><![CDATA[Luis O.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Hinojosa]]></surname>
<given-names><![CDATA[Carlos A.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Instituto Nacional de Ciencias Médicas y Nutrición Salvador Zubirán Section of Vascular Surgery and Endovascular Therapy Department of Surgery]]></institution>
<addr-line><![CDATA[Mexico City ]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Instituto Nacional de Cancerología Traslational Medicine Laboratory ]]></institution>
<addr-line><![CDATA[Mexico City ]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Instituto Nacional de Cancerología Laboratory of Biological Cancer-causing agents ]]></institution>
<addr-line><![CDATA[Mexico City ]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af4">
<institution><![CDATA[,Instituto Nacional de Ciencias Médicas y Nutrición Salvador Zubirán Biochemistry Unit ]]></institution>
<addr-line><![CDATA[Mexico City ]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>03</month>
<year>2022</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>03</month>
<year>2022</year>
</pub-date>
<volume>50</volume>
<numero>1</numero>
<fpage>26</fpage>
<lpage>31</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S2696-130X2022000100026&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S2696-130X2022000100026&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S2696-130X2022000100026&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract  Background: The arteriovenous fistula (AVF) is the first option as vascular access for patients with end-stage renal disease. The main cause of failure is stenosis of the venous portion of the AVF secondary to intimal hyperplasia.  Material and methods: The objective of this study was to evaluate the relative expression of miR-145, miR-146, and miR-155. We used an experimental model of intimal hyperplasia creating an AVF. We included six Wistar Rats. The analysis and validation of miR-145, miR-146, and miR-155 was performed using real-time polymerase chain reaction.  Results: There is a down-regulation of miR-145 in the vein (0.233 ± 0.3, p = 0.0030) and artery (0.33 ± 0.4, p = 0.0630), compared to the control. Likewise, we found upregulation of miR-146 in the vein (29 ± 0.25, p = 0.0256) and artery (34.66 ± 0.35, p = 0.0345) compared to the control and upregulation of miR-155 in the vein (20 ± 0.39, p = 0.0231) and artery (22.66 ± 0.49, p = 0.0234) compared to the control.  Conclusions: We found alterations in the expression of miR-145, miR-146, and miR-155 comparing the fistula versus control, both in the arterial and venous portions in an experimental model of AVF.]]></p></abstract>
<abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen  Antecedentes: La fístula arteriovenosa es la primera opción como acceso vascular para pacientes con enfermedad renal terminal. La principal causa de fracaso es la estenosis de la porción venosa de la fístula secundaria a hiperplasia intimal.  Material y métodos: El objetivo de este estudio fue evaluar la expresión relativa de miR-145, miR-146 y miR-155. Utilizamos un modelo experimental de hiperplasia intimal realizando una fístula arteriovenosa. Incluimos 6 ratas Wistar. El análisis y validación de miR-145, miR-146 y miR-155 se realizó mediante reacción en cadena de la polimerasa en tiempo real.  Resultados: Hay una regulación a la baja de miR-145 en la vena (0.233 ± 0.3, p = 0.0030) y arteria (0.33 ± 0.4, p = 0.0630), en comparación con el control. Asimismo, encontramos regulación positiva de miR-146 en vena (29 ± 0.25, p = 0.0256) y arteria (34.66 ± 0.35, p = 0.0345) en comparación con el control y regulación positiva de miR-155 en vena (20 ± 0.39, p = 0.0231) y arteria (22.66 ± 0.49, p = 0.0234) en comparación con el control.  Conclusiones: Encontramos alteraciones en la expresión de miR-145, miR-146 y miR-155 comparando la fístula vs. control, tanto en la porción arterial como venosa en un modelo experimental de fistula.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Hyperplasia]]></kwd>
<kwd lng="en"><![CDATA[Tunica intima]]></kwd>
<kwd lng="en"><![CDATA[MicroRNAs]]></kwd>
<kwd lng="en"><![CDATA[Myocites]]></kwd>
<kwd lng="en"><![CDATA[Smooth muscle]]></kwd>
<kwd lng="en"><![CDATA[Arteriovenous fistula]]></kwd>
<kwd lng="es"><![CDATA[Hiperplasia]]></kwd>
<kwd lng="es"><![CDATA[Túnica íntima]]></kwd>
<kwd lng="es"><![CDATA[MicroRNA]]></kwd>
<kwd lng="es"><![CDATA[Miocitos]]></kwd>
<kwd lng="es"><![CDATA[Músculo liso]]></kwd>
<kwd lng="es"><![CDATA[Fístula arteriovenosa]]></kwd>
</kwd-group>
</article-meta>
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