<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2524-177X</journal-id>
<journal-title><![CDATA[Revista médica del Hospital General de México]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. med. Hosp. Gen. Méx.]]></abbrev-journal-title>
<issn>2524-177X</issn>
<publisher>
<publisher-name><![CDATA[Sociedad Médica del Hospital General de México A.C.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2524-177X2024000400004</article-id>
<article-id pub-id-type="doi">10.24875/hgmx.23000061</article-id>
<title-group>
<article-title xml:lang="en"><![CDATA[Cell death process associated with the renin-angiotensin-aldosterone system]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Gómez-Montañez]]></surname>
<given-names><![CDATA[Emiliano]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Rojas-Salazar]]></surname>
<given-names><![CDATA[Yareli L.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Sánchez-Mendoza]]></surname>
<given-names><![CDATA[Alicia]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Sánchez-Aguilar]]></surname>
<given-names><![CDATA[María]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Autónoma de Ciudad Juárez Institute of Biomedical Sciences ]]></institution>
<addr-line><![CDATA[Ciudad Juárez Chihuahua]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Secretaría de Salud Instituto Nacional de Cardiología Ignacio Chávez Laboratory of Vascular Reactivity, Department of Pharmacology Dr. Rafael Méndez]]></institution>
<addr-line><![CDATA[Mexico City ]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2024</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2024</year>
</pub-date>
<volume>87</volume>
<numero>4</numero>
<fpage>175</fpage>
<lpage>183</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S2524-177X2024000400004&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S2524-177X2024000400004&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S2524-177X2024000400004&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract The renin-angiotensin-aldosterone system (RAAS) is a crucial regulator of blood pressure and fluid balance. This metabolic pathway plays an important role in the genesis of cardiovascular and renal diseases. This review explores the relationship between RAAS activation and cell death processes such as apoptosis, necrosis, necroptosis, ferroptosis, and autophagy. Therefore, a literature search was carried out in PubMed, Science Direct, Scielo, and Google Scholar databases. Articles from the last years (2017-2022) were selected using keywords such as “RAAS”, “Angiotensin II”, “Cell death”, and/or “Kidney injury”.  Conclusion: The RAAS, known mainly for its cardiovascular and renal roles, also plays a significant role in cell death processes. Understanding these connections may lead to new therapeutic strategies for the management of cardiovascular and renal diseases.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Renin-angiotensin system]]></kwd>
<kwd lng="en"><![CDATA[Death cell]]></kwd>
<kwd lng="en"><![CDATA[Hypertension]]></kwd>
<kwd lng="en"><![CDATA[Apoptosis]]></kwd>
</kwd-group>
</article-meta>
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