<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2448-8909</journal-id>
<journal-title><![CDATA[Medicina crítica (Colegio Mexicano de Medicina Crítica)]]></journal-title>
<abbrev-journal-title><![CDATA[Med. crít. (Col. Mex. Med. Crít.)]]></abbrev-journal-title>
<issn>2448-8909</issn>
<publisher>
<publisher-name><![CDATA[Colegio Mexicano de Medicina Crítica A.C.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2448-89092024000700615</article-id>
<article-id pub-id-type="doi">10.35366/119538</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Cetoacidosis diabética euglucémica en un paciente con choque séptico secundario a infección por Rickettsia typhi: a propósito de un caso]]></article-title>
<article-title xml:lang="en"><![CDATA[Euglycemic diabetic ketoacidosis in a patient with septic shock secondary to infection by Rickettsia typhi: a case report]]></article-title>
<article-title xml:lang="pt"><![CDATA[Cetoacidose diabética euglicêmica em paciente com choque séptico secundário à infecção por Rickettsia typhi: relato de caso]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Unzueta Ortiz]]></surname>
<given-names><![CDATA[Daniel]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Hernández Gutiérrez]]></surname>
<given-names><![CDATA[Mariel Alejandra]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Díaz Mendoza]]></surname>
<given-names><![CDATA[Jesús Elías]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Corral González]]></surname>
<given-names><![CDATA[Julio César]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Hospital General de Chihuahua «Dr. Salvador Zubirán Anchondo»  ]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>México</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>00</month>
<year>2024</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>00</month>
<year>2024</year>
</pub-date>
<volume>38</volume>
<numero>7</numero>
<fpage>615</fpage>
<lpage>620</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S2448-89092024000700615&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S2448-89092024000700615&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S2448-89092024000700615&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen: La rickettsiosis es una entidad clínica poco frecuente en las unidades de cuidados intensivos en México, su prevalencia aumenta en los estados del norte del país, principalmente Chihuahua y Sonora. Las garrapatas y piojos son considerados los principales vectores hematófagos causantes de rickettsiosis en estos estados, probablemente asociado con las características climatológicas. La falta de signos y/o síntomas específicos en etapas tempranas de la enfermedad dificulta el diagnóstico y tratamiento oportuno, lo que conlleva a una tasa de mortalidad elevada debido al rápido desarrollo de choque séptico y subsecuente a síndrome de disfunción orgánica múltiple. Un número importante de pacientes que desarrolla choque séptico presenta hiperglucemia debido al estrés metabólico y resistencia a la insulina. La tendencia actual de tratamiento con uso de inhibidores del cotransportador sodio-glucosa tipo 2 (SGLT-2) ha ido en aumento debido a sus efectos benéficos cardiovasculares, raramente al asociarse con estados de estrés metabólicos pueden generar descompensación del equilibrio ácido-base sin alterar los niveles de glicemia capilar (cetoacidosis diabética euglucémica). Por lo que el tratamiento debe estar enfocado en resolver el proceso desencadenante junto con la corrección del equilibrio ácido-base. Presentamos el caso de una mujer en la quinta década de la vida con antecedente de diabetes mellitus tipo 2 en tratamiento con iSGLT-2 y con exposición a animales domésticos infestados por piojos y garrapatas, desarrollando clínica compatible con rickettsiosis y datos de disfunción orgánica, iniciando tratamiento con antibiótico, durante su evolución presenta cetoacidosis diabética euglucémica asociado con uso de dapagliflozina.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract: Rickettsiosis is an infrequent clinical entity in intensive care units in Mexico, its prevalence increases in the northern states of the country, mainly Chihuahua and Sonora. Ticks and lice are considered the main hematophagous vectors causing rickettsiosis in these states, probably associated with the climatological characteristics. The lack of specific signs and/or symptoms in early stages of the disease makes diagnosis and timely treatment difficult, leading to a high mortality rate due to the rapid development of septic shock and subsequent multiple organ dysfunction syndrome. A significant number of patients who develop septic shock present hyperglycemia due to metabolic stress, insulin resistance. The current trend of treatment with sodium-glucose cotransporter type 2 (SGLT-2) inhibitors has been increasing due to their beneficial cardiovascular effects, rarely when associated with metabolic stress states can generate decompensation of the acid-base balance without altering capillary glycemia levels (euglycemic diabetic ketoacidosis). Therefore, treatment should be focused on resolving the triggering process together with the correction of the acid-base balance. We present the case of a female patient in the fifth decade of life with a history of type 2 diabetes mellitus under treatment with (iSGLT-2) and with exposure to domestic animals.]]></p></abstract>
<abstract abstract-type="short" xml:lang="pt"><p><![CDATA[Resumo: A rickettsiose é uma entidade clínica rara em unidades de terapias intensivas no México, a sua prevalência aumenta nos estados do norte do país, principalmente Chihuahua e Sonora. Carrapatos e piolhos são considerados os principais vetores hematófagos causadores de Rickettsioses nesses estados, provavelmente associados a características climatológicas. A falta de sinais e/ou sintomas específicos nas fases iniciais da doença dificulta o diagnóstico e o tratamento oportunos, o que leva a uma elevada taxa de mortalidade devido ao rápido desenvolvimento de choque séptico e subsequente síndrome de disfunção de múltiplos órgãos. Um número significativo de pacientes que desenvolve choque séptico apresenta hiperglicemia devido ao estresse metabólico e resistência à insulina. A tendência atual de tratamento com uso de inibidores do cotransportador sódio-glicose 2 (SGLT-2) vem aumentando devido aos seus efeitos cardiovasculares benéficos, raramente, quando associados a estados de estresse metabólico, podem gerar descompensação do equilíbrio ácido-base sem alteração dos níveis de glicose no sangue capilar (cetoacidose diabética euglicêmica). Portanto, o tratamento deve ser focado na resolução do processo desencadeante e na correção do equilíbrio ácido-base. Apresentamos o caso de uma paciente do sexo feminino, na quinta década de vida, com histórico de diabetes mellitus tipo 2 em tratamento com (ISGLT-2) e com exposição a animais domésticos infestados por piolhos e carrapatos, desenvolvendo sintomas compatíveis com riquetsioses e sinais de disfunção orgânica, iniciando tratamento com antibióticos, durante sua evolução apresentou cetoacidose diabética euglicêmica associada ao uso de dapagliflozina.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[rickettsiosis]]></kwd>
<kwd lng="es"><![CDATA[iSGLT-2]]></kwd>
<kwd lng="es"><![CDATA[cetoacidosis diabética euglucémica]]></kwd>
<kwd lng="es"><![CDATA[Rickettsia typhi]]></kwd>
<kwd lng="en"><![CDATA[rickettsiosis]]></kwd>
<kwd lng="en"><![CDATA[iSGLT-2]]></kwd>
<kwd lng="en"><![CDATA[euglycemic diabetic ketoacidosis]]></kwd>
<kwd lng="en"><![CDATA[Rickettsia typhi]]></kwd>
<kwd lng="pt"><![CDATA[rickettsiose]]></kwd>
<kwd lng="pt"><![CDATA[ISGLT-2]]></kwd>
<kwd lng="pt"><![CDATA[cetoacidose diabética euglicêmica]]></kwd>
<kwd lng="pt"><![CDATA[Rickettssia typhi]]></kwd>
</kwd-group>
</article-meta>
</front><back>
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