<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2448-8909</journal-id>
<journal-title><![CDATA[Medicina crítica (Colegio Mexicano de Medicina Crítica)]]></journal-title>
<abbrev-journal-title><![CDATA[Med. crít. (Col. Mex. Med. Crít.)]]></abbrev-journal-title>
<issn>2448-8909</issn>
<publisher>
<publisher-name><![CDATA[Colegio Mexicano de Medicina Crítica A.C.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2448-89092024000600520</article-id>
<article-id pub-id-type="doi">10.35366/119242</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Inmunosupresión e hipercoagulabilidad secundaria a deficiencia de proteína S en paciente críticamente enfermo]]></article-title>
<article-title xml:lang="en"><![CDATA[Immunosuppression and hypercoagulability due to protein S deficiency in critically ill patient]]></article-title>
<article-title xml:lang="pt"><![CDATA[Imunossupressão e hipercoagulabilidade secundária à deficiência proteína S em paciente em estado crítico]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Giraldo Sánchez]]></surname>
<given-names><![CDATA[Javier Mauricio]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Giraldo Jaramillo]]></surname>
<given-names><![CDATA[Valentina]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Giraldo Jaramillo]]></surname>
<given-names><![CDATA[Juliana]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Tolima UCI Honda  ]]></institution>
<addr-line><![CDATA[Honda Tolima]]></addr-line>
<country>Colombia</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad de la Sabana  ]]></institution>
<addr-line><![CDATA[Bogotá ]]></addr-line>
<country>Colombia</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>00</month>
<year>2024</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>00</month>
<year>2024</year>
</pub-date>
<volume>38</volume>
<numero>6</numero>
<fpage>520</fpage>
<lpage>523</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S2448-89092024000600520&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S2448-89092024000600520&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S2448-89092024000600520&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen: Las deficiencias de glicoproteínas endógenas anticoagulantes se encuentran en el espectro clínico de enfermedades derivadas de deleciones cromosomales o mutaciones en la secuencia aminoacídica de su conformación, generando impacto negativo no sólo en su actividad, sino también en su funcionalidad, lo que precipita eventos trombóticos y cuadros de inmunosupresión. Sin embargo, existe un grupo de pacientes que, sin tener trastornos autosómicos dominantes, presentan deficiencias de este tipo de glicoproteínas. Con base en dicho análisis, presentamos el caso clínico de un adulto mayor con alta carga de enfermedad y morbilidad relacionada con la deficiencia descrita, con el objeto de permitir al clínico el discernimiento en escenarios clínicos específicos.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract: Deficiencies of endogenous anticoagulant glycoproteins are found in the clinical spectrum of disease derived from chromosomal deletions or mutations in the amino acid sequence of their conformation, generating a negative impact not only on their activity but also on their functionality, precipitating thrombotic events and immunosuppression symptoms. However, there is a group of patients who without having autosomal dominant disorders, have deficiencies of this type of glycoprotein.]]></p></abstract>
<abstract abstract-type="short" xml:lang="pt"><p><![CDATA[Resumo: Deficiências de glicoproteínas endógenas anticoagulantes são encontradas no espectro clínico de doenças derivadas de deleções ou mutações cromossômicas na sequência de aminoácidos de sua conformação, gerando impacto negativo não apenas em sua atividade, mas também em sua funcionalidade, precipitando eventos trombóticos e imunossupressão. Porém, existe um grupo de pacientes que, sem apresentar doenças autossômica dominantes, apresentam deficiências desse tipo de glicoproteínas. Com base nesta análise, apresentamos o caso clínico de um idoso com elevada carga da doença e morbidade relacionada à deficiência descrita, a fim de permitir ao clínico discernir em cenários clínicos específicos.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[hipercoagulabilidad]]></kwd>
<kwd lng="es"><![CDATA[inmunosupresión]]></kwd>
<kwd lng="es"><![CDATA[glicoproteínas]]></kwd>
<kwd lng="es"><![CDATA[cirrosis]]></kwd>
<kwd lng="es"><![CDATA[falla cardiaca]]></kwd>
<kwd lng="en"><![CDATA[hypercoagulability]]></kwd>
<kwd lng="en"><![CDATA[immunosuppression]]></kwd>
<kwd lng="en"><![CDATA[glycoproteins]]></kwd>
<kwd lng="en"><![CDATA[cirrhosis]]></kwd>
<kwd lng="en"><![CDATA[cardiac failure]]></kwd>
<kwd lng="pt"><![CDATA[hipercoagulabilidade]]></kwd>
<kwd lng="pt"><![CDATA[imunossupressão]]></kwd>
<kwd lng="pt"><![CDATA[glicoproteínas]]></kwd>
<kwd lng="pt"><![CDATA[cirrose]]></kwd>
<kwd lng="pt"><![CDATA[insuficiência cardíaca]]></kwd>
</kwd-group>
</article-meta>
</front><back>
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