<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2007-1124</journal-id>
<journal-title><![CDATA[Revista mexicana de ciencias pecuarias]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. mex. de cienc. pecuarias]]></abbrev-journal-title>
<issn>2007-1124</issn>
<publisher>
<publisher-name><![CDATA[Instituto Nacional de Investigaciones Forestales, Agrícolas y Pecuarias]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2007-11242024000100192</article-id>
<article-id pub-id-type="doi">10.22319/rmcp.v15i1.6463</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Análisis in silico de genes diana de miRNAs posiblemente inducidos por la infección con tuberculosis]]></article-title>
<article-title xml:lang="en"><![CDATA[In silico analysis of miRNA target genes possibly induced by tuberculosis infection]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Rodríguez-Hernández]]></surname>
<given-names><![CDATA[Elba]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Quintas-Granados]]></surname>
<given-names><![CDATA[Laura Itzel]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Milian Suazo]]></surname>
<given-names><![CDATA[Feliciano]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Anaya Escalera]]></surname>
<given-names><![CDATA[Ana María]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Instituto Nacional de Investigaciones Forestales Agrícolas y Pecuarias Centro Nacional de Investigación Disciplinaria en Fisiología y Mejoramiento Animal ]]></institution>
<addr-line><![CDATA[ Querétaro]]></addr-line>
<country>México</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad Autónoma de la Ciudad de México  ]]></institution>
<addr-line><![CDATA[ Ciudad de México]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Universidad Autónoma de Querétaro  ]]></institution>
<addr-line><![CDATA[ Querétaro]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>03</month>
<year>2024</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>03</month>
<year>2024</year>
</pub-date>
<volume>15</volume>
<numero>1</numero>
<fpage>192</fpage>
<lpage>207</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S2007-11242024000100192&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S2007-11242024000100192&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S2007-11242024000100192&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen El objetivo fue identificar mediante análisis in silico los genes a los cuales se unen los miR-146a, miR-146b y miR-155 y, analizar las rutas metabólicas en las que intervienen durante la infección con tuberculosis. Para el análisis se utilizó: miRBase, UniProtKB, TargetScan Human, miRDB y miRTarBase. El miR-146a interacciona o se une a genes importantes en la adhesión celular y el proceso de fagocitosis (CLDN16 y ATP6V1C2, respectivamente) (P, 0.05), esta interacción podría tener implicaciones importantes en la patogénesis de la tuberculosis o enfermedades relacionadas. Los resultados de este trabajo sugieren que la activación de mecanismos moleculares específicos en respuesta a la tuberculosis está regulada por los miR-146a, miR-146b y miR-155. Los genes con los cuales los miR-146a y miR-155 interaccionan o se unen, están involucrados en la respuesta inmune y en procesos celulares imprescindibles durante una infección por tuberculosis.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract The objective was to identify, through in silico analysis, the genes to which miR-146a, miR-146b, and miR-155 bind and to analyze the metabolic pathways in which they participate during tuberculosis infection. For the analysis, it was used: miRBase, UniProtKB, TargetScan Human, miRDB, and miRTarBase. miR-146a interacts with or binds to genes important in cell adhesion and the process of phagocytosis (CLDN16 and ATP6V1C2, respectively) (P&lt; 0.05); this interaction could have important implications in the pathogenesis of tuberculosis or related diseases. The results of this work suggest that the activation of specific molecular mechanisms in response to tuberculosis is regulated by miR-146a, miR-146b, and miR-155. The genes with which miR-146a and miR-155 interact or bind are involved in the immune response and cellular processes essential during tuberculosis infection.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Mycobacterium]]></kwd>
<kwd lng="es"><![CDATA[miR-146a]]></kwd>
<kwd lng="es"><![CDATA[miR-146b]]></kwd>
<kwd lng="es"><![CDATA[miR-155]]></kwd>
<kwd lng="es"><![CDATA[Predicción]]></kwd>
<kwd lng="es"><![CDATA[Diagnóstico]]></kwd>
<kwd lng="en"><![CDATA[Mycobacterium]]></kwd>
<kwd lng="en"><![CDATA[miR-146a]]></kwd>
<kwd lng="en"><![CDATA[miR-146b]]></kwd>
<kwd lng="en"><![CDATA[miR-155]]></kwd>
<kwd lng="en"><![CDATA[Prediction]]></kwd>
<kwd lng="en"><![CDATA[Diagnosis]]></kwd>
</kwd-group>
</article-meta>
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