<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1870-7203</journal-id>
<journal-title><![CDATA[Acta médica Grupo Ángeles]]></journal-title>
<abbrev-journal-title><![CDATA[Acta méd. Grupo Ángeles]]></abbrev-journal-title>
<issn>1870-7203</issn>
<publisher>
<publisher-name><![CDATA[Grupo Ángeles, Servicios de Salud]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1870-72032025000100052</article-id>
<article-id pub-id-type="doi">10.35366/119354</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Una nueva propuesta: diabetes tipo 3, una revisión narrativa]]></article-title>
<article-title xml:lang="en"><![CDATA[A new appraisal: type 3 diabetes, a narrative review]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Córdova Pluma]]></surname>
<given-names><![CDATA[Víctor Huggo]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
<xref ref-type="aff" rid="A a"/>
<xref ref-type="aff" rid="A1"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Zabaneh Campos]]></surname>
<given-names><![CDATA[Víctor]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Rodríguez Weber]]></surname>
<given-names><![CDATA[Federico Leopoldo]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Cabrera Jardines]]></surname>
<given-names><![CDATA[Ricardo]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[González Araiza]]></surname>
<given-names><![CDATA[Rodrigo]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Cantú Gómez]]></surname>
<given-names><![CDATA[Amanda]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Gutiérrez Buenabad]]></surname>
<given-names><![CDATA[Diana]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Aguilar Domingo]]></surname>
<given-names><![CDATA[Moisés]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Hospital Angeles México Unidad de Neurociencias ]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>México</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Hospital Angeles Pedregal Departamento de Medicina Interna ]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>México</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Hospital Angeles Pedregal Universidad Anáhuac México ]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>México</country>
</aff>
<aff id="Af4">
<institution><![CDATA[,Universidad La Salle  ]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af5">
<institution><![CDATA[,Clínica Plexus-NBT  ]]></institution>
<addr-line><![CDATA[Guadalajara Jalisco]]></addr-line>
<country>México</country>
</aff>
<aff id="Af6">
<institution><![CDATA[,Universidad de Manchester  ]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>United Kingdom</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>03</month>
<year>2025</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>03</month>
<year>2025</year>
</pub-date>
<volume>23</volume>
<numero>1</numero>
<fpage>52</fpage>
<lpage>58</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S1870-72032025000100052&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S1870-72032025000100052&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S1870-72032025000100052&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen: Este artículo de revisión explora la relación entre la resistencia a la insulina en el sistema nervioso central (SNC) y su impacto en enfermedades neurodegenerativas, particularmente en el espectro de la diabetes tipo 2 y el espectro de la enfermedad de Alzheimer. A través de un análisis de vías moleculares clave, como PI3K/Akt, AMPK y JNK, el artículo describe cómo estas alteraciones metabólicas afectan la neuroplasticidad y promueven el daño neuronal. También aborda el papel de la disfunción mitocondrial y los estados proinflamatorios crónicos, que crean entornos de estrés oxidativo y contribuyen a la acumulación de proteínas tóxicas, acelerando la neurodegeneración. Además, el artículo examina métodos diagnósticos emergentes, incluyendo biomarcadores plasmáticos y de LCR, qEEG, fMRI y MRS, que permiten la identificación temprana de alteraciones en la conectividad y cambios metabólicos en el cerebro. Estos métodos ofrecen oportunidades para la detección e intervención tempranas en etapas iniciales, con importantes implicaciones clínicas y preventivas. Finalmente, se discuten intervenciones basadas en el estilo de vida y posibles enfoques terapéuticos, como la insulina intranasal y los moduladores de la inflamación, para frenar la progresión del deterioro cognitivo y mejorar la calidad de vida en pacientes de alto riesgo.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract: This review article explores the relationship between insulin resistance in the central nervous system (CNS) and its impact on neurodegenerative diseases, particularly in type 2 diabetes and Alzheimer&#8217;s disease spectrum. Through an analysis of key molecular pathways involved, such as PI3K/Akt, AMPK, and JNK, the article describes how these metabolic alterations impair neuroplasticity and promote neuronal damage. It also addresses the role of mitochondrial dysfunction and chronic proinflammatory states, which create oxidative stress environments that contribute to the buildup of toxic proteins, accelerating neurodegeneration. Additionally, the article examines emerging diagnostic methods including plasma and CSF biomarkers, qEEG, fMRI, and MRS, which allow early identification of connectivity alterations and metabolic changes in the brain. These methods provide early detection and intervention opportunities in initial stages, with significant clinical and preventive implications. Finally, lifestyle-based interventions and potential therapeutic approaches, such as intranasal insulin and inflammation modulators, are discussed to slow cognitive decline progression and improve the quality of life in high-risk patients.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[diabetes tipo 3]]></kwd>
<kwd lng="es"><![CDATA[disfunción mitocondrial]]></kwd>
<kwd lng="es"><![CDATA[Alzheimer]]></kwd>
<kwd lng="es"><![CDATA[resistencia a la insulina]]></kwd>
<kwd lng="es"><![CDATA[demencia]]></kwd>
<kwd lng="en"><![CDATA[diabetes type 3]]></kwd>
<kwd lng="en"><![CDATA[mitochondrial dysfunction]]></kwd>
<kwd lng="en"><![CDATA[Alzheimer&#8217;s disease]]></kwd>
<kwd lng="en"><![CDATA[insulin resistance]]></kwd>
<kwd lng="en"><![CDATA[dementia]]></kwd>
</kwd-group>
</article-meta>
</front><back>
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