<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1870-199X</journal-id>
<journal-title><![CDATA[Revista odontológica mexicana]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. Odont. Mex]]></abbrev-journal-title>
<issn>1870-199X</issn>
<publisher>
<publisher-name><![CDATA[Universidad Nacional Autónoma de México, Facultad de Odontología]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1870-199X2018000300137</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Análisis de la relación entre la hipomineralización incisivo molar y los factores asociados a su etiología]]></article-title>
<article-title xml:lang="en"><![CDATA[Analysis of the relationship between molar incisor hypomineralization and the factors associated with its etiology]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Gómez Clavel]]></surname>
<given-names><![CDATA[José Francisco]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Amato Martínez]]></surname>
<given-names><![CDATA[Dante]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Trejo Iriarte]]></surname>
<given-names><![CDATA[Cynthia Georgina]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[García Muñoz]]></surname>
<given-names><![CDATA[Alejandro]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Nacional Autónoma de México FES ]]></institution>
<addr-line><![CDATA[Iztacala ]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad Nacional Autónoma de México FES Departamento de Medicina]]></institution>
<addr-line><![CDATA[Iztacala ]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Universidad Nacional Autónoma de México FES ]]></institution>
<addr-line><![CDATA[Iztacala ]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>09</month>
<year>2018</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>09</month>
<year>2018</year>
</pub-date>
<volume>22</volume>
<numero>3</numero>
<fpage>137</fpage>
<lpage>143</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S1870-199X2018000300137&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S1870-199X2018000300137&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S1870-199X2018000300137&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[RESUMEN El objetivo de este artículo es analizar la relación entre la hipomineralización incisivo molar (HIM) y los factores asociados a su etiología publicados en la literatura.  Material y métodos: El estudio se basó en una búsqueda de estudios epidemiológicos de casos y controles de HIM que describieron un factor etiológico asociado para obtener las odds ratio (OR) necesarias para analizar la prevalencia del factor afectado y su posible papel en la etiología de la condición.  Resultados: Ocho artículos cumplieron los criterios para el análisis. La población total analizada consistió en una muestra de 7,901 sujetos, de los cuales 992 tenían HIM. En estos sujetos fueron reportados como factor asociado (FA) para HIM: asma (OR = 4.4954), uso de antibióticos (OR = 5.5348), fiebre (OR = 4.0545) y neumonía.  Conclusión: Los resultados del presente estudio sugieren que un FA común a todos los casos de HIM estudiados es un proceso inflamatorio que conduce a una mayor concentración de agentes en el microambiente en el que se desarrollan las células formadoras del esmalte, lo que aumenta la presencia de proteínas en la matriz del esmalte o interfieren con su hidrólisis y eliminación y produce como resultado una deficiente mineralización.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[ABSTRACT Hypomineralization enamel of the first permanent molars is the most common developmental abnormalities observed in the teeth. The aetiology of MIH remains unclear and may have a multifactor aetiology. The aim of this paper is to analyze the relationship between MIH and associated factors published in the literature.  Material and methods: The study was based on a search for epidemiological case-control studies of MIH that described an associated etiological factor, in order to obtain the odds ratios needed to analyze the prevalence of the factor concerned and its possible role in the etiology of the condition.  Results: The initial search produced 50 articles, eight of which met the criteria for the analysis. The total population analyzed consisted of a sample of 7,901 subjects, 992 of whom had MIH (i.e., a prevalence of 12.55%). Asthma was reported as an etiological factor in five papers, which included 474 subjects with MIH with an OR of 4.4954 (p &lt; 0.0001). Antibiotic use was reported as an etiological factor in three papers, which reported on a population of 231 subjects with MIH and OR of 5.5348 (p &lt; 0.0001). Fever was reported as an etiological factor in two papers, involving a population of 176 subjects with MIH and an OR of 4.0545 (p &lt; 0.0001). Pneumonia was reported as an etiological factor in two papers, which dealt with a population of 454 cases of MIH and produced an OR of 2.285 (p &lt; 0.0001).  Conclusion: The results of the present study suggest that one etiological factor common to all of the MIH cases studied is an infl ammatory process, in which the presence of agents that cause alterations in ameloblasts can lead to higher concentrations of these agents in the microenvironment in which enamel forming cells develop, thus increasing the presence of proteins in the enamel matrix or interfering with their hydrolysis and removal, producing defects in enamel mineralization.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Etiología]]></kwd>
<kwd lng="es"><![CDATA[hipomineralización incisivo molar (HIM)]]></kwd>
<kwd lng="es"><![CDATA[odds ratios]]></kwd>
<kwd lng="es"><![CDATA[defectos del esmalte]]></kwd>
<kwd lng="en"><![CDATA[Etiology]]></kwd>
<kwd lng="en"><![CDATA[molar incisor hypomineralization (MIH)]]></kwd>
<kwd lng="en"><![CDATA[odds ratios]]></kwd>
<kwd lng="en"><![CDATA[enamel defects]]></kwd>
</kwd-group>
</article-meta>
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