<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1665-5044</journal-id>
<journal-title><![CDATA[Revista mexicana de neurociencia]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. mex. neurocienc.]]></abbrev-journal-title>
<issn>1665-5044</issn>
<publisher>
<publisher-name><![CDATA[Academia Mexicana de Neurología A.C.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1665-50442026000200066</article-id>
<article-id pub-id-type="doi">10.24875/rmn.25000045</article-id>
<title-group>
<article-title xml:lang="en"><![CDATA[Diabetic striatopathy in Hispanic patients: recognizing a variable yet treatable movement disorder]]></article-title>
<article-title xml:lang="es"><![CDATA[Estriatopatía diabética en pacientes hispanos: reconociendo un trastorno del movimiento variable pero tratable]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Díaz-Garza]]></surname>
<given-names><![CDATA[Carlos A.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[López-Díaz]]></surname>
<given-names><![CDATA[Verónica I.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Santoyo-Fexas]]></surname>
<given-names><![CDATA[Leticia A.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Luna-Rangel]]></surname>
<given-names><![CDATA[Francisco A.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Martínez-Ramirez]]></surname>
<given-names><![CDATA[Daniel]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Instituto Tecnológico y de Estudios Superiores de Monterrey Escuela de Medicina y Ciencias de la Salud ]]></institution>
<addr-line><![CDATA[Monterrey ]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>04</month>
<year>2026</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>04</month>
<year>2026</year>
</pub-date>
<volume>27</volume>
<numero>2</numero>
<fpage>66</fpage>
<lpage>71</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S1665-50442026000200066&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S1665-50442026000200066&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S1665-50442026000200066&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract  Objective To describe clinical features, diagnostic challenges, and therapeutic response of diabetic striatopathy in Latin American patients, an underrepresented population in current literature, and to highlight the importance of early recognition and interdisciplinary collaboration in improving diagnosis and management.  Methods Case series of two Latin American women with poorly controlled type 2 diabetes mellitus who developed hyperkinetic movement disorders. Clinical presentation, neuroimaging findings, diagnostic approach, and clinical outcomes following metabolic and symptomatic treatment were analyzed.  Results One patient presented with acute-onset chorea accompanied by pain and hypoesthesia, with striatal hyperdensity on computed tomography and T1 hyperintensity on magnetic resonance imaging, achieving complete symptom resolution within 48 hours after intensive insulin therapy. The second patient developed progressive choreiform movements, initially misdiagnosed as epilepsy, without response to antiepileptic drugs. Neuroimaging confirmed diabetic striatopathy; insulin therapy produced partial improvement, and adjunctive haloperidol reduced movement severity, with residual symptoms at discharge. Both cases demonstrated characteristic imaging findings despite distinct clinical phenotypes.  Conclusions Diabetic striatopathy shows heterogeneous clinical presentations and may mimic other neurological disorders, leading to diagnostic delays. Early recognition and prompt glycemic control are essential, although treatment response varies and may require individualized management strategies.]]></p></abstract>
<abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen  Objetivo Describir las características clínicas, los desafíos diagnósticos y la respuesta terapéutica de la estriatopatía diabética en pacientes latinoamericanos, una población escasamente representada en la literatura, y resaltar la necesidad de reconocimiento clínico temprano y colaboración interdisciplinaria.  Métodos Serie de casos de dos mujeres latinoamericanas con diabetes mellitus tipo 2 mal controlada que desarrollaron trastornos del movimiento hipercinéticos. Se analizaron las manifestaciones clínicas, hallazgos de neuroimagen, abordaje diagnóstico y evolución clínica tras tratamiento metabólico y sintomático.  Resultados Una paciente presentó corea de inicio agudo asociada a dolor e hipoestesia, con hiperdensidad estriatal en tomografía e hiperintensidad en T1 en resonancia magnética, logrando resolución completa tras control glucémico intensivo con insulina en 48 horas. La segunda paciente desarrolló movimientos coreiformes progresivos, inicialmente diagnosticados como epilepsia, sin respuesta a antiepilépticos. La neuroimagen confirmó estriatopatía diabética; el tratamiento con insulina produjo mejoría parcial y la adición de haloperidol redujo la severidad de los movimientos, persistiendo síntomas residuales. Ambos casos mostraron hallazgos radiológicos característicos pese a fenotipos clínicos distintos.  Conclusiones La estriatopatía diabética presenta manifestaciones clínicas heterogéneas y puede simular otras enfermedades neurológicas, favoreciendo retrasos diagnósticos. El reconocimiento temprano y el control glucémico oportuno son fundamentales, aunque la respuesta terapéutica puede variar y requerir manejo individualizado.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Diabetes]]></kwd>
<kwd lng="en"><![CDATA[Hyperglycemia]]></kwd>
<kwd lng="en"><![CDATA[Chorea]]></kwd>
<kwd lng="en"><![CDATA[Basal ganglia]]></kwd>
<kwd lng="en"><![CDATA[Case report]]></kwd>
<kwd lng="es"><![CDATA[Diabetes]]></kwd>
<kwd lng="es"><![CDATA[Hiperglucemia]]></kwd>
<kwd lng="es"><![CDATA[Corea]]></kwd>
<kwd lng="es"><![CDATA[Ganglios basales]]></kwd>
<kwd lng="es"><![CDATA[Reporte de caso]]></kwd>
</kwd-group>
</article-meta>
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