<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1665-5044</journal-id>
<journal-title><![CDATA[Revista mexicana de neurociencia]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. mex. neurocienc.]]></abbrev-journal-title>
<issn>1665-5044</issn>
<publisher>
<publisher-name><![CDATA[Academia Mexicana de Neurología A.C.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1665-50442019000300162</article-id>
<article-id pub-id-type="doi">10.24875/rmn.19000010</article-id>
<title-group>
<article-title xml:lang="en"><![CDATA[Immunology of Alzheimer&#8217;s disease]]></article-title>
<article-title xml:lang="es"><![CDATA[Inmunología de la enfermedad de Alzheimer]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Sosa-García]]></surname>
<given-names><![CDATA[Lucila I.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Hernández-Jiménez]]></surname>
<given-names><![CDATA[Janeth]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Moral-Huerta]]></surname>
<given-names><![CDATA[Diana I. Del]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Duyckaerts]]></surname>
<given-names><![CDATA[Charles]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Calderón-Garcidueñas]]></surname>
<given-names><![CDATA[Ana L.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Veracruzana Institute of Biomedical Research Doctoral Program in Biomedical Sciences]]></institution>
<addr-line><![CDATA[Xalapa ]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Hôpital Universitaire Pitié-Salpêtrière Departament of Neuropathology ]]></institution>
<addr-line><![CDATA[Paris ]]></addr-line>
<country>France</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Universidad Veracruzana Faculty of Medicine ]]></institution>
<addr-line><![CDATA[ Veracruz]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>06</month>
<year>2019</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>06</month>
<year>2019</year>
</pub-date>
<volume>20</volume>
<numero>3</numero>
<fpage>162</fpage>
<lpage>170</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S1665-50442019000300162&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S1665-50442019000300162&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S1665-50442019000300162&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Alzheimer&#8217;s disease (AD) is a multifactorial neurodegenerative pathology. Neuroinflammation is an early event of the presymptomatic stages in AD and contributes to its progression. We review the participation of astrocytes, microglia and blood-brain barrier cells, the mechanisms of cell death and the inflammatory factors such as chemokines, interferons and Toll-like receptors involved in progression and perpetuation of AD. Some of its prognostic and therapeutic possibilities are also mentioned. Identifying the different actors involved in inflammation and the main mechanisms of damage might allow the development of preventive strategies and treatments to fight against this devastating disease.]]></p></abstract>
<abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen La enfermedad de Alzheimer (EA) es una patología neurodegenerativa multifactorial. La neuroinflamación es un evento temprano de las etapas presintomáticas en la EA y contribuye a su progresión. En este trabajo revisamos la participación de astrocitos, microglia y células de la barrera hematoencefálica, los mecanismos de muerte celular y los factores inflamatorios como las quimiocinas, los interferones y los receptores tipo Toll que participan en la progresión y la perpetuación de esta enfermedad. También se mencionan algunas de sus posibilidades pronósticas y terapéuticas. La identificación de los diferentes actores involucrados en la inflamación y de los principales mecanismos de daño podrían permitir el desarrollo de estrategias y tratamientos preventivos para combatir esta enfermedad devastadora.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Immunology]]></kwd>
<kwd lng="en"><![CDATA[Alzheimer&#8217;s disease]]></kwd>
<kwd lng="en"><![CDATA[Microglia]]></kwd>
<kwd lng="en"><![CDATA[Innate immunity]]></kwd>
<kwd lng="en"><![CDATA[Lymphocytes]]></kwd>
<kwd lng="es"><![CDATA[Inmunología]]></kwd>
<kwd lng="es"><![CDATA[Enfermedad de Alzheimer]]></kwd>
<kwd lng="es"><![CDATA[Microglia]]></kwd>
<kwd lng="es"><![CDATA[Inmunidad innata]]></kwd>
<kwd lng="es"><![CDATA[Linfocitos]]></kwd>
</kwd-group>
</article-meta>
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