<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1665-1146</journal-id>
<journal-title><![CDATA[Boletín médico del Hospital Infantil de México]]></journal-title>
<abbrev-journal-title><![CDATA[Bol. Med. Hosp. Infant. Mex.]]></abbrev-journal-title>
<issn>1665-1146</issn>
<publisher>
<publisher-name><![CDATA[Instituto Nacional de Salud, Hospital Infantil de México Federico Gómez]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1665-11462021000300200</article-id>
<article-id pub-id-type="doi">10.24875/bmhim.20000108</article-id>
<title-group>
<article-title xml:lang="en"><![CDATA[Placental SLC38A4 gene polymorphisms 1304 G &gt; A and 292 C &gt; T, and their association with glucose &gt; 95 mg/dL in normal weight full-term healthy newborns]]></article-title>
<article-title xml:lang="es"><![CDATA[Polimorfismos 1304 G &gt; A y 292 C &gt; T del gen placentario SLC38A4 y su asociación con glucosa &gt; 95 mg/dL en recién nacidos a término, sanos y con peso normal al nacimiento]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Galaviz-Hernández]]></surname>
<given-names><![CDATA[Carlos]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Sosa-Macías]]></surname>
<given-names><![CDATA[Martha]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Rodríguez-Morán]]></surname>
<given-names><![CDATA[Martha]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Martínez-Aguilar]]></surname>
<given-names><![CDATA[Gerardo]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Guerrero-Romero]]></surname>
<given-names><![CDATA[Fernando]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[González-Rentería]]></surname>
<given-names><![CDATA[Siblie M.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Instituto Politécnico Nacional Centro Interdisciplinario de Investigación para el Desarrollo Integral Regional Unidad Durango Academia de Genómica]]></institution>
<addr-line><![CDATA[Durango ]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Instituto Mexicano del Seguro Social Unidad de Investigación Biomédica ]]></institution>
<addr-line><![CDATA[Durango ]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Universidad Juárez del Estado de Durango Facultad de Ciencias Químicas ]]></institution>
<addr-line><![CDATA[Durango ]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>06</month>
<year>2021</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>06</month>
<year>2021</year>
</pub-date>
<volume>78</volume>
<numero>3</numero>
<fpage>200</fpage>
<lpage>206</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S1665-11462021000300200&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S1665-11462021000300200&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S1665-11462021000300200&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract  Background: The SLC38A4 gene encodes for the SNAT4 protein, which has been related to glucose metabolic alterations in human newborns. This study aimed to determine whether the 1304 G &gt; A and 292 C &gt; T polymorphisms of the SLC38A4 gene are associated with the presence of glucose levels &gt; 95 mg/dL in normal weight full-term healthy newborns.  Methods: We conducted a case–control study and analyzed 50 normal weight full-term healthy newborns. Groups were defined based on glucose levels: &gt; 95 mg/dL (cases; n = 13) and &lt; 95 mg/dL (controls; n = 37). The 1304 G &gt; A and 292 C &gt; T polymorphisms of the SLC38A4 gene were determined through quantitative polymerase chain reaction using placental DNA. The association between polymorphism and glucose levels &gt; 95 mg/dL was established using multivariate logistic regression analysis.  Results: No significant differences were observed either for gestational age or body weight at birth between groups. In the case group, newborns showed significantly higher homeostatic model assessment for insulin resistance than those in the control group (p &lt; 0.0005). The odds ratio (OR) between the SLC38A4 gene 292 C &gt; T single-nucleotide polymorphism (SNP) and glucose levels &gt; 95 mg/dL was 7.78 (p = 0.024), whereas no significant association was found for the 1304 G &gt; A SNP (OR 1.46; p = 0.77).  Conclusions: Our results suggest that the SLC38A4 gene 292 C &gt; T SNP is associated with glucose levels &gt; 95 mg/dL in normal weight full-term healthy newborns.]]></p></abstract>
<abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen  Introducción: El gen SLC38A4 codifica la proteína SNAT4, que se ha relacionado con alteraciones en el metabolismo de la glucosa en los humanos. El objetivo de este estudio fue determinar si los polimorfismos 1304 G &gt; A y 292 C &gt; T del gen SLC38A4 se asocian con concentraciones de glucosa &gt; 95 mg/dL en recién nacidos a término  Métodos: Se llevó a cabo un estudio de casos y controles con 50 recién nacidos a término, sanos, con peso normal al nacimiento. Los grupos se definieron de acuerdo con las concentraciones de glucosa: &gt; 95 mg/dL (casos; n = 13) y &lt; 95 mg/dL (controles; n = 37). Los polimorfismos 1304 G &gt; A y 292 C &gt; T del gen SLC38A4 se genotipificaron por qPCR utilizando ADN de la placenta. La asociación entre los polimorfismos y la concentración de glucosa &gt; 95 mg/dL se estableció mediante la estimación de la razón de momios (RM) en un análisis múltiple de regresión logística.  Resultados: No se observaron diferencias estadísticamente significativas para la edad gestacional y el peso al nacer entre los grupos de estudio. El modelo homeostático para evaluar la resistencia a la insulina (HOMA-IR) fue significativamente más alto en los recién nacidos del grupo de casos que en el grupo control (p &lt; 0.0005). La RM mostró asociación significativa entre el polimorfismo de nucleótido único (SNP) 292 C &gt; T del gen SLC38A4 y la concentración de glucosa &gt; 95 mg/dL (RM: 7.78; p = 0.024); el SNP 1304 G &gt; A no mostró asociación significativa (RM: 1.46; p = 0.77).  Conclusiones: Los resultados de este estudio sugieren que el SNP 292 C &gt; T del gen SLC38A4 se asocia con concentraciones de glucosa &gt; 95 mg/dL en recién nacidos a término.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[SLC38A4]]></kwd>
<kwd lng="en"><![CDATA[Polymorphisms]]></kwd>
<kwd lng="en"><![CDATA[Glucose]]></kwd>
<kwd lng="en"><![CDATA[Newborns]]></kwd>
<kwd lng="es"><![CDATA[SLC38A4]]></kwd>
<kwd lng="es"><![CDATA[Polimorfismos]]></kwd>
<kwd lng="es"><![CDATA[Glucosa]]></kwd>
<kwd lng="es"><![CDATA[Recién nacidos]]></kwd>
</kwd-group>
</article-meta>
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