<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1405-888X</journal-id>
<journal-title><![CDATA[TIP. Revista especializada en ciencias químico-biológicas]]></journal-title>
<abbrev-journal-title><![CDATA[TIP]]></abbrev-journal-title>
<issn>1405-888X</issn>
<publisher>
<publisher-name><![CDATA[Universidad Nacional Autónoma de México, Facultad de Estudios Superiores Zaragoza]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1405-888X2024000100007</article-id>
<article-id pub-id-type="doi">10.22201/fesz.23958723e.2024.684</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[El papel de la ferroptosis en la enfermedad renal diabética]]></article-title>
<article-title xml:lang="en"><![CDATA[The role of ferroptosis in diabetic kidney disease]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Peña-Montes]]></surname>
<given-names><![CDATA[Donovan J.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Saavedra-Molina]]></surname>
<given-names><![CDATA[Alfredo]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Michoacana de San Nicolás de Hidalgo Instituto de Investigaciones Químico Biológicas Laboratorio de Bioquímica]]></institution>
<addr-line><![CDATA[Morelia Michoacán]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>00</month>
<year>2024</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>00</month>
<year>2024</year>
</pub-date>
<volume>27</volume>
<fpage>0</fpage>
<lpage>0</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S1405-888X2024000100007&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S1405-888X2024000100007&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S1405-888X2024000100007&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen La enfermedad renal diabética (ERD) es una complicación grave de la diabetes y la principal causa de enfermedad renal terminal en México y el mundo. Esta revisión examina su compleja fisiopatología y se destaca el papel emergente de la ferroptosis, una forma de muerte celular regulada y desencadenada por la descoordinación metabólica del hierro, de los tioles y de los lípidos. La hiperglucemia crónica, característica de la diabetes, da lugar a una serie de eventos que incluyen el estrés oxidativo, la inflamación y las alteraciones metabólicas, creando un ambiente propicio para el desarrollo de la ferroptosis en el tejido renal. La evidencia reciente sugiere que la desregulación del metabolismo del hierro y la peroxidación lipídica exacerbada, son elementos clave para su aparición y que contribuyen significativamente al daño renal en la ERD. También se analizó la interacción entre los mecanismos tradicionales del daño renal diabético y la ferroptosis y cómo esta forma de muerte celular podría ser un nuevo objetivo de búsqueda terapéutica para ayudar al desarrollo de estrategias innovadoras de prevención y tratamiento de esta complicación.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Diabetic kidney disease (DKD) is a severe complication of diabetes and the leading cause of end-stage renal disease in Mexico and worldwide. This review examines the complex pathophysiology of DKD, highlighting the emerging role of ferroptosis, an iron-dependent form of regulated cell death. Chronic hyperglycemia, characteristic of diabetes, triggers a cascade of events including oxidative stress, inflammation, and metabolic alterations, creating an environment conducive to ferroptosis in renal tissue. Recent evidence suggests that dysregulation of iron metabolism and exacerbated lipid peroxidation, key elements of ferroptosis, significantly contribute to renal damage in DKD. This review analyzes the interaction between traditional mechanisms of diabetic kidney injury and ferroptosis, exploring how this form of cell death could represent a new therapeutic target. Understanding the role of ferroptosis in DKD is crucial for developing innovative strategies for the prevention and treatment of this complication.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[diabetes]]></kwd>
<kwd lng="es"><![CDATA[enfermedad renal diabética]]></kwd>
<kwd lng="es"><![CDATA[hierro]]></kwd>
<kwd lng="es"><![CDATA[ferroptosis]]></kwd>
<kwd lng="es"><![CDATA[muerte celular]]></kwd>
<kwd lng="en"><![CDATA[diabetes]]></kwd>
<kwd lng="en"><![CDATA[diabetic kidney disease]]></kwd>
<kwd lng="en"><![CDATA[iron]]></kwd>
<kwd lng="en"><![CDATA[ferroptosis]]></kwd>
<kwd lng="en"><![CDATA[cell death]]></kwd>
</kwd-group>
</article-meta>
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