<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1405-888X</journal-id>
<journal-title><![CDATA[TIP. Revista especializada en ciencias químico-biológicas]]></journal-title>
<abbrev-journal-title><![CDATA[TIP]]></abbrev-journal-title>
<issn>1405-888X</issn>
<publisher>
<publisher-name><![CDATA[Universidad Nacional Autónoma de México, Facultad de Estudios Superiores Zaragoza]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1405-888X2020000100205</article-id>
<article-id pub-id-type="doi">10.22201/fesz.23958723e.2020.0.225</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[La plasticidad del hepatocito y su relevancia en la fisiología y la patología hepática]]></article-title>
<article-title xml:lang="en"><![CDATA[Hepatocyte plasticity and its relevance in liver physiology and pathology]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Ríos-López]]></surname>
<given-names><![CDATA[Diana G.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Aranda-López]]></surname>
<given-names><![CDATA[Yuli]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Sosa-Garrocho]]></surname>
<given-names><![CDATA[Marcela]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Macías-Silva]]></surname>
<given-names><![CDATA[Marina]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Nacional Autónoma de México Departamento de Biología Celular y del Desarrollo Instituto de Fisiología Celular]]></institution>
<addr-line><![CDATA[Ciudad de México ]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>00</month>
<year>2020</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>00</month>
<year>2020</year>
</pub-date>
<volume>23</volume>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S1405-888X2020000100205&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S1405-888X2020000100205&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S1405-888X2020000100205&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen El hígado es uno de los principales órganos encargados de mantener la homeostasis en vertebrados, además de poseer una gran capacidad regenerativa. El hígado está constituido por diversos tipos celulares que de forma coordinada contribuyen para que el órgano funcione eficientemente. Los hepatocitos representan el tipo celular principal de este órgano y llevan a cabo la mayoría de sus actividades; además, constituyen una población heterogénea de células epiteliales con funciones especializadas en el metabolismo. El fenotipo de los hepatocitos está controlado por diferentes vías de señalización, como la vía del TGF&#946;/Smads, la ruta Hippo/YAP-TAZ y la vía Wnt/&#946;-catenina, entre otras. Los hepatocitos son células que se encuentran normalmente en un estado quiescente, aunque cuentan con una plasticidad intrínseca que se manifiesta en respuesta a diversos daños en el hígado; así, estas células reactivan su capacidad proliferativa o cambian su fenotipo a través de procesos celulares como la transdiferenciación o la transformación, para contribuir a mantener la homeostasis del órgano en condiciones saludables o desarrollar diversas patologías.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract The liver is one of the most important organs that contribute to maintain metabolic homeostasis in vertebrates, with a high regenerative capacity. Liver is constituted by diverse cellular types, which work together to accomplish the organ function. Hepatocytes are the main cellular type responsible for most of hepatic functions; they are a heterogeneous population with specialized metabolic functions. Several signaling pathways such as TGF-&#946;/Smads, Hippo/YAP-TAZ and Wnt/ &#946;-catenin, among others, regulate hepatocytes phenotype. These cells are normally in a quiescent state but they exhibit plasticity in response to liver damage; thus, they can proliferate or change their phenotype through process such as transdifferentiation or transformation in order to contribute to the maintenance of liver homeostasis or during the development of diverse pathologies.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[plasticidad celular]]></kwd>
<kwd lng="es"><![CDATA[hepatocito]]></kwd>
<kwd lng="es"><![CDATA[hígado]]></kwd>
<kwd lng="es"><![CDATA[transdiferenciación]]></kwd>
<kwd lng="es"><![CDATA[transformación]]></kwd>
<kwd lng="es"><![CDATA[EMT]]></kwd>
<kwd lng="en"><![CDATA[liver]]></kwd>
<kwd lng="en"><![CDATA[hepatocyte]]></kwd>
<kwd lng="en"><![CDATA[plasticity]]></kwd>
<kwd lng="en"><![CDATA[transdifferentiation]]></kwd>
<kwd lng="en"><![CDATA[transformation]]></kwd>
<kwd lng="en"><![CDATA[EMT]]></kwd>
</kwd-group>
</article-meta>
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