<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1405-888X</journal-id>
<journal-title><![CDATA[TIP. Revista especializada en ciencias químico-biológicas]]></journal-title>
<abbrev-journal-title><![CDATA[TIP]]></abbrev-journal-title>
<issn>1405-888X</issn>
<publisher>
<publisher-name><![CDATA[Universidad Nacional Autónoma de México, Facultad de Estudios Superiores Zaragoza]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1405-888X2005000200071</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Relación de la estructura de los receptores NMDA con su función en la retina]]></article-title>
<article-title xml:lang="en"><![CDATA[Structure-function relationship of NMDA receptors in the retina]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Lee-Rivera]]></surname>
<given-names><![CDATA[Irene]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[López-Colomé]]></surname>
<given-names><![CDATA[Ana María]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,UNAM Instituto de Fisiología Celular ]]></institution>
<addr-line><![CDATA[México, D.F. ]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>00</month>
<year>2005</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>00</month>
<year>2005</year>
</pub-date>
<volume>8</volume>
<numero>2</numero>
<fpage>71</fpage>
<lpage>81</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S1405-888X2005000200071&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S1405-888X2005000200071&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S1405-888X2005000200071&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen La función del glutamato en la retina, como neurotransmisor, factor trófico o neurotoxina, se relaciona directamente con la diversidad y la composición heteromérica de sus receptores. Los receptores de glutamato de tipo NMDA (NMDARs) son tetrámeros integrados por dos subunidades NR1, que forman un canal iónico permeable al calcio, y subunidades NR2 y NR3, que modulan su actividad. Los NMDARs de la retina incluyen subunidades NR2, así como variantes postranscripcionales de NR1 específicas, tanto en el tejido diferenciado como el embrionario, cuya estructura determina diferencias funcionales significativas entre el cerebro y la retina. Consecuentemente, fármacos que protegen a las neuronas de la excitotoxicidad del glutamato en el cerebro, no tienen efecto en la retina. La repercusiónclínica de estos resultados es evidente, ya que permitirá, en el futuro, el diseño de compuestos protectores de la muerte neuronal en la retina en padecimientos relacionados con la elevación de la concentración extracelular del glutamato, como el glaucoma, la oclusión vascular, la neuropatía óptica y la isquemia, entre otros.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract The multiple actions of glutamate in the retina, as a neurotransmitter, a trophic factor or a neurotoxin, are directly related to the heteromeric composition of the NMDA type of glutamate receptors present in this tissue. The tetrameric NMDARs include two NR1 subunits which form the calcium-permeable cation channel, in addition to NR2 and NR3 subunits which modulate its activity. Recent results demonstrate that retinal NMDARs might include tissue-specific NR1 splice variants and NR2 subunits, in both mature and embryonic tissue, which determine structural and functional differences with brain receptors. As a consequence, drugs shown to protect brain neurons from glutamate-induced neuronal death, are ineffective in the retina. The clarification of the precise subunit composition of NMDARs in the retina will allow the future design of drugs aimed to prevent neuronal loss in pathological conditions such as glaucoma, vascular occlusion, optic neuropathy and ischemia, in which glutamate excitotoxicity seems to be involved.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Desarrollo embrionario]]></kwd>
<kwd lng="es"><![CDATA[neurotransmisión]]></kwd>
<kwd lng="es"><![CDATA[NR1]]></kwd>
<kwd lng="es"><![CDATA[NR2]]></kwd>
<kwd lng="es"><![CDATA[receptor de glutamato]]></kwd>
<kwd lng="en"><![CDATA[Embryonic development]]></kwd>
<kwd lng="en"><![CDATA[neurotransmission]]></kwd>
<kwd lng="en"><![CDATA[NR1]]></kwd>
<kwd lng="en"><![CDATA[NR2]]></kwd>
<kwd lng="en"><![CDATA[glutamate receptors]]></kwd>
</kwd-group>
</article-meta>
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