<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0188-6266</journal-id>
<journal-title><![CDATA[Acta universitaria]]></journal-title>
<abbrev-journal-title><![CDATA[Acta univ]]></abbrev-journal-title>
<issn>0188-6266</issn>
<publisher>
<publisher-name><![CDATA[Universidad de Guanajuato, Dirección de Investigación y Posgrado]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0188-62662017000600078</article-id>
<article-id pub-id-type="doi">10.15174/au.2017.1346</article-id>
<title-group>
<article-title xml:lang="en"><![CDATA[Oxidative damage in lipids in the central nervous system and spleen in iron-deficient mice]]></article-title>
<article-title xml:lang="es"><![CDATA[Daño oxidativo en lípidos del sistea nervioso central y bazo de ratones con deficiencia de Hierro]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Martínez Domínguez]]></surname>
<given-names><![CDATA[Anabella]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Contreras]]></surname>
<given-names><![CDATA[Irazú]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Estrada]]></surname>
<given-names><![CDATA[José Antonio]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Autónoma del Estado de México Facultad de Medicina Laboratorio de Neuroquímica]]></institution>
<addr-line><![CDATA[Toluca Estado de México]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2017</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2017</year>
</pub-date>
<volume>27</volume>
<numero>6</numero>
<fpage>78</fpage>
<lpage>83</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S0188-62662017000600078&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S0188-62662017000600078&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S0188-62662017000600078&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="en"><p><![CDATA[ABSTRACT Oxidative stress (OS) is considered a risk factor for neurodegenerative disorders. Iron concentrations have been related to OS; however, the effect of iron deficiency (ID) on the induction of OS in the central nervous system (CNS) is unknown. A murine model of chronic diet-induced ID using 2 month-old male BALB/c mice (6 specimen), was used to determine its role in OS induction. Lipid peroxidation was determined by measuring thiobarbituric acid-reactive species (TBARS) in the CNS, using the spleen for comparison. A decrease of peroxidation products in the CNS of the ID group (ID CNS), compared to controls fed on a regular diet, was found. The spleen showed a significant increase of lipid peroxidation in the ID group. Our results suggest that chronic ID may have differential effects on OS in the CNS and peripheral tissues. A decreased amount of reactive oxygen species in the basal state may be related to alterations on normal CNS metabolism and functions under chronic ID.]]></p></abstract>
<abstract abstract-type="short" xml:lang="es"><p><![CDATA[RESUMEN El estrés oxidativo (EO) es factor de riesgo en trastornos neurodegenerativos. Las concentraciones de hierro se han relacionado al EO; sin embargo, el efecto de la deficiencia de hierro (DH) en la inducción de EO en el Sistema Nervioso Central (SNC) es desconocido. Se empleó un modelo murino consistente de 6 machos BALB/c de dos meses de edad para DH crónica inducida por dieta, para determinar su papel en la inducción de EO. La peroxidación lipídica se determinó midiendo especies reactivas al ácido tiobarbitúrico (TBARS) en el SNC de ratones de 2 meses de edad, usando el bazo para comparación. Se encontró una disminución en productos de peroxidación en el SNC DH, comparado con controles alimentados con dieta normal. El bazo mostró un aumento significativo de la peroxidación lipídica en el grupo DH. Los resultados sugieren que la DH crónica puede tener efectos diferenciales en el SNC y tejidos periféricos. Una cantidad reducida de especies reactivas de oxigeno en estado basal podría estar relacionada con alteraciones en el metabolismo y funcionamiento normal del SNC bajo condiciones de DH crónica.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Oxidative stress]]></kwd>
<kwd lng="en"><![CDATA[lipid peroxidation]]></kwd>
<kwd lng="en"><![CDATA[iron deficiency]]></kwd>
<kwd lng="en"><![CDATA[central nervous system]]></kwd>
<kwd lng="en"><![CDATA[spleen]]></kwd>
<kwd lng="es"><![CDATA[Estrés oxidativo]]></kwd>
<kwd lng="es"><![CDATA[peroxidación lipídica]]></kwd>
<kwd lng="es"><![CDATA[deficiencia de hierro]]></kwd>
<kwd lng="es"><![CDATA[sistema nervioso central]]></kwd>
<kwd lng="es"><![CDATA[bazo]]></kwd>
</kwd-group>
</article-meta>
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