<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0187-5337</journal-id>
<journal-title><![CDATA[Perinatología y reproducción humana]]></journal-title>
<abbrev-journal-title><![CDATA[Perinatol. Reprod. Hum.]]></abbrev-journal-title>
<issn>0187-5337</issn>
<publisher>
<publisher-name><![CDATA[Instituto Nacional de Perinatología]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0187-53372024000200004</article-id>
<article-id pub-id-type="doi">10.24875/per.24000007</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Generalidades del antígeno leucocitario humano G y su relación con la pérdida gestacional recurrente]]></article-title>
<article-title xml:lang="en"><![CDATA[Generalities of the human leukocyte antigen G and its relationship with recurrent pregnancy loss]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Villagómez-Sánchez]]></surname>
<given-names><![CDATA[Pamela A.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Suárez-Souto]]></surname>
<given-names><![CDATA[Ma. Antonieta]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Sánchez-Monroy]]></surname>
<given-names><![CDATA[Virginia]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Ciudad de México  ]]></institution>
<addr-line><![CDATA[Ciudad de México ]]></addr-line>
<country>México</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Instituto Politécnico Nacional Escuela Superior de Medicina ]]></institution>
<addr-line><![CDATA[Ciudad de México ]]></addr-line>
<country>México</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>08</month>
<year>2024</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>08</month>
<year>2024</year>
</pub-date>
<volume>38</volume>
<numero>2</numero>
<fpage>53</fpage>
<lpage>59</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S0187-53372024000200004&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S0187-53372024000200004&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S0187-53372024000200004&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen La pérdida recurrente de la gestación (RPL) se define como la pérdida gestacional en dos o más ocasiones en forma consecutiva o alterna. Se conoce como una patología multifactorial y compleja en la cual más del 50% de los casos son de causa idiopática. Una de las causas vinculadas a RPL y que se ha venido estudiando es el antígeno leucocitario humano G (HLA-G), que es una molécula codificada por el complejo principal de histocompatibilidad, la cual actúa con otras moléculas y células inmunológicas, principalmente linfocitos NK, inhibiendo su función citotóxica sobre el trofoblasto promoviendo el proceso de implantación y placentación del feto. El objetivo es revisar la literatura de RPL asociada a la molécula HLA-G. Los resultados de las investigaciones han descrito que la expresión de la molécula HLA-G en el trofoblasto y su molécula soluble (sHLA-G) son bajas en pacientes con RPL, respecto a las que no presentan RPL. Se han documentado además polimorfismos en el receptor que se acopla HLA-G y en la propia molécula que también se han asociado a RPL, sugiriendo disminución en su acoplamiento, que lleva a la célula NK a ejercer su función citotóxica, provocando la terminación de la gestación.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Recurrent pregnancy loss (RPL) is defined as pregnancy loss on two or more occasions consecutively or alternately. It is known as a multifactorial and complex pathology where more than 50% of cases have an idiopathic cause. One of the causes linked to RPL and that has been studied is the human leukocyte antigen G (HLA-G), which is a molecule encoded by the major histocompatibility complex, which acts with other molecules and immune cells, mainly NK lymphocytes, inhibiting their cytotoxic function on the trophoblast, promoting the process of implantation and placentation of the fetus. The objective is to review the literature on RPL associated with the HLA-G molecule. Research results have described that the expression of the HLA-G molecule in the trophoblast and its soluble molecule (sHLA-G) are low in patients with RPL, compared to those who do not present RPL. Polymorphisms have also been documented in the receptor that engages HLA-G and in the molecule itself that have also been associated with RPL, suggesting a decrease in its coupling, which leads the NK cell to exert its cytotoxic function, causing the termination of the gestation.]]></p></abstract>
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<kwd lng="es"><![CDATA[Embarazo]]></kwd>
<kwd lng="es"><![CDATA[Tolerancia inmunológica]]></kwd>
<kwd lng="es"><![CDATA[HLA-G]]></kwd>
<kwd lng="es"><![CDATA[Pérdida gestacional recurrente]]></kwd>
<kwd lng="es"><![CDATA[Aborto recurrente]]></kwd>
<kwd lng="en"><![CDATA[Pregnancy]]></kwd>
<kwd lng="en"><![CDATA[Immunological tolerance]]></kwd>
<kwd lng="en"><![CDATA[HLA-G]]></kwd>
<kwd lng="en"><![CDATA[Recurrent pregnancy loss]]></kwd>
<kwd lng="en"><![CDATA[Recurrent abortion]]></kwd>
</kwd-group>
</article-meta>
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