<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0016-3813</journal-id>
<journal-title><![CDATA[Gaceta médica de México]]></journal-title>
<abbrev-journal-title><![CDATA[Gac. Méd. Méx]]></abbrev-journal-title>
<issn>0016-3813</issn>
<publisher>
<publisher-name><![CDATA[Academia Nacional de Medicina de México A.C.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0016-38132024000500002</article-id>
<article-id pub-id-type="doi">10.24875/gmm.24000125</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Arilaminas N-acetiltransferasas 1 y 2: fisiología, genética y epigenética]]></article-title>
<article-title xml:lang="en"><![CDATA[Arylamines N-acetyltransferases 1 and 2: physiology, genetics and epigenetics]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Hernández-González]]></surname>
<given-names><![CDATA[Oswaldo]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Martínez-Leija]]></surname>
<given-names><![CDATA[Miguel E.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Paz-Rodríguez]]></surname>
<given-names><![CDATA[Víctor A.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Portales-Pérez]]></surname>
<given-names><![CDATA[Diana P.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Facultad de Ciencias Químicas, Universidad Autónoma de San Luis Potosí, San Luis Potosí Laboratorio de Inmunología y Biología Celular y Molecular ]]></institution>
<addr-line><![CDATA[San Luis Potosí ]]></addr-line>
<country>México</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>10</month>
<year>2024</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>10</month>
<year>2024</year>
</pub-date>
<volume>160</volume>
<numero>5</numero>
<fpage>498</fpage>
<lpage>504</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S0016-38132024000500002&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S0016-38132024000500002&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S0016-38132024000500002&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen Las arilaminas N-acetiltransferasas NAT1 y NAT2 son enzimas principalmente conocidas por su participación en la fase II del proceso de detoxificación. Ambas enzimas se localizan en células inmunes y en diversos órganos como hígado, intestino, pulmón y piel. Estudios recientes sugieren su posible papel en el proceso de carcinogénesis o metabolismo celular. La actividad catalítica por su alta influencia en la concentración celular de su cofactor acetilcoenzima A puede participar en múltiples procesos celulares, dependiente de su concentración citoplasmática. Se realizó una búsqueda en las bases de datos ENCODE y PubMed para identificar información de interés sobre la epigenética de los promotores de NAT1 y NAT2, así como reportes sobre los posibles mecanismos implicados en las enfermedades de estudio. El efecto de NAT1 en el metabolismo del folato ha llevado a proponerlo como un posible regulador epigenético.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Arylamine N-Acetyltransferases NAT1 and NAT2 are enzymes mainly known for participating in phase II of the detoxification process. Both enzymes are present in various organs, such as the liver, intestine, lung, skin, and immune cells. Recent studies implicate its possible role in carcinogenesis or cell metabolism; The catalytic activity, representing a substantial influence on the cellular concentration of its Acetyl Coenzyme A cofactor, can influence multiple cellular processes, depending on its cytoplasmic concentration. A review was carried out in the databases ENCODE and PubMed to identify information of interest on the epigenetics of NAT1 and NAT2 promoters and reports proposing the possible mechanisms involved in the diseases under study. The effect of NAT1 on folate metabolism has led to it being proposed as a possible epigenetic regulator.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[ARNm]]></kwd>
<kwd lng="es"><![CDATA[Epigenética]]></kwd>
<kwd lng="es"><![CDATA[NAT1]]></kwd>
<kwd lng="es"><![CDATA[NAT2]]></kwd>
<kwd lng="es"><![CDATA[Polimorfismo]]></kwd>
<kwd lng="en"><![CDATA[mRNA]]></kwd>
<kwd lng="en"><![CDATA[Epigenetics]]></kwd>
<kwd lng="en"><![CDATA[NAT1]]></kwd>
<kwd lng="en"><![CDATA[NAT2]]></kwd>
<kwd lng="en"><![CDATA[Polymorphism]]></kwd>
</kwd-group>
</article-meta>
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