<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2448-9190</journal-id>
<journal-title><![CDATA[Revista alergia México]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. alerg. Méx.]]></abbrev-journal-title>
<issn>2448-9190</issn>
<publisher>
<publisher-name><![CDATA[Colegio Mexicano de Inmunología Clínica y Alergia A.C.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2448-91902018000200160</article-id>
<article-id pub-id-type="doi">10.29262/ram.v65i2.359</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Sueño y sistema inmune]]></article-title>
<article-title xml:lang="en"><![CDATA[Sleep and immune system]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Rico-Rosillo]]></surname>
<given-names><![CDATA[María Guadalupe]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Vega-Robledo]]></surname>
<given-names><![CDATA[Gloria Bertha]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Nacional Autónoma de México Facultad de Medicina Unidad de Medicina Experimental]]></institution>
<addr-line><![CDATA[ Ciudad de México]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>06</month>
<year>2018</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>06</month>
<year>2018</year>
</pub-date>
<volume>65</volume>
<numero>2</numero>
<fpage>160</fpage>
<lpage>170</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S2448-91902018000200160&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S2448-91902018000200160&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S2448-91902018000200160&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen: El sueño es un proceso que ocupa la tercera parte de la vida del ser humano y resulta imprescindible para que el individuo mantenga la homeostasis del organismo. Emerge como un regulador importante del sistema inmune, ya que durante el sueño se llevan a cabo las funciones necesarias para mantener su equilibrio. Por otro lado, la reducción de sueño tiene efectos adversos que alteran el metabolismo y produce incremento en la secreción de la proteína C reactiva, interleucina (IL)-6 y factor de necrosis tumoral (TNF). Estas citocinas activan a NF-&#954;B, por lo que la alteración en el sueño puede ser un factor de riesgo para desarrollar enfermedades inflamatorias crónicas y metabólicas. Las citocinas proinflamatorias IL-1, IL-6 y TNF aumentan el sueño de movimientos oculares no rápidos y las antiinflamatorias como IL-4 e IL-10 lo disminuyen. El sueño puede modificar la función del sistema inmune induciendo cambios en el eje hipotálamo-pituitaria-adrenal y el sistema nervioso simpático. A su vez, el ritmo circadiano de hormonas como el cortisol y la adrenalina, que descienden en la noche, favorece diferentes actividades del sistema inmune. El objetivo de la presente revisión es abordar diversos aspectos del sueño y su relación con el sistema inmune.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract: Sleep is a process that occupies one third part of the life of the human being, and it is essential in order for the individual to be able to maintain body homeostasis. It emerges as an important regulator of the immune system since, during sleep, the necessary functions to maintain its balance are carried out. On the other hand, decreased sleep has deleterious effects that alter the metabolism and produce an increase in the secretion of C-reactive protein, interleukin (IL)-6 and tumor necrosis factor (TNF). These cytokines activate NF-&#954;B; therefore, sleep disturbance can be a risk factor for the development of chronic inflammatory and metabolic diseases. Pro-inflammatory cytokines IL-1, IL-6 and TNF increase non-rapid eye movement sleep, whereas anti-inflammatory cytokines such as IL-4 and IL-10 decrease it. Sleep can modify the immune system function by inducing changes in the hypothalamus-pituitary-adrenal axis and the sympathetic nervous system. In turn, the circadian rhythm of hormones such as cortisol and adrenaline, which have a nocturnal decrease, favors different activities of the immune system. The purpose of the present review is to address different aspects of sleep and their relationship with the immune system.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Sueño]]></kwd>
<kwd lng="es"><![CDATA[Privación del sueño]]></kwd>
<kwd lng="es"><![CDATA[Citocinas]]></kwd>
<kwd lng="es"><![CDATA[Sistema inmune]]></kwd>
<kwd lng="es"><![CDATA[Inflamación]]></kwd>
<kwd lng="en"><![CDATA[Sleep]]></kwd>
<kwd lng="en"><![CDATA[Sleep deprivation]]></kwd>
<kwd lng="en"><![CDATA[Cytokines]]></kwd>
<kwd lng="en"><![CDATA[Immune system]]></kwd>
<kwd lng="en"><![CDATA[Inflammation]]></kwd>
</kwd-group>
</article-meta>
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<given-names><![CDATA[JA]]></given-names>
</name>
</person-group>
<article-title xml:lang=""><![CDATA[Comparing circadian dynamics in primary derived stem cells from different sources of human adult tissue]]></article-title>
<source><![CDATA[Stem Cells Int]]></source>
<year>2017</year>
</nlm-citation>
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</back>
</article>
