<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2448-8909</journal-id>
<journal-title><![CDATA[Medicina crítica (Colegio Mexicano de Medicina Crítica)]]></journal-title>
<abbrev-journal-title><![CDATA[Med. crít. (Col. Mex. Med. Crít.)]]></abbrev-journal-title>
<issn>2448-8909</issn>
<publisher>
<publisher-name><![CDATA[Colegio Mexicano de Medicina Crítica A.C.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2448-89092021000300154</article-id>
<article-id pub-id-type="doi">10.35366/100005</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Síndrome de Fisher-Evans asociado a pancreatitis aguda]]></article-title>
<article-title xml:lang="en"><![CDATA[Fisher-Evans syndrome associated to acute pancreatitis]]></article-title>
<article-title xml:lang="pt"><![CDATA[Síndrome de Fisher-Evans associada a pancreatite aguda]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Domínguez-Pérez]]></surname>
<given-names><![CDATA[Rafael]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Arrazola-García]]></surname>
<given-names><![CDATA[Fernando Valentín]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,IMSS Hospital General Regional No. 1 «Dr. Carlos Mac Gregor Sánchez Navarro» Unidad de Terapia Intensiva]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>06</month>
<year>2021</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>06</month>
<year>2021</year>
</pub-date>
<volume>35</volume>
<numero>3</numero>
<fpage>154</fpage>
<lpage>156</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S2448-89092021000300154&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S2448-89092021000300154&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S2448-89092021000300154&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen: El síndrome de Fisher-Evans o de Evans es la manifestación de anemia hemolítica inmunitaria que puede ser simultánea o subsecuente a trombocitopenia inmunitaria. Debido a que su diagnóstico no es común, en la actualidad, no hay cifras exactas acerca de su prevalencia. De acuerdo con su origen, puede ser un evento idiopático o secundario. Presentamos el caso de un hombre de 58 años que desarrolló síndrome de Fisher-Evans asociado a pancreatitis aguda, en la Unidad de Terapia Intensiva (UTI). Caracterizado por anemia hemolítica inmunitaria (Coombs positivo) y trombocitopenia; además presentó disfunción renal. Durante su estancia hospitalaria, recibió tratamiento con esteroide; metilprednisolona intravenosa por tres días, prednisona vía oral (1 mg/kg/día) e inmunoglobulina humana (0.4 g/kg/día) por cinco días.  Resultados:  Presentó mejoría del cuadro clínico, con resolución de la anemia y trombocitopenia. Fue valorado por el Servicio de Nefrología; que instauró manejo con hemodiálisis y realizó biopsia renal con reporte de necrosis tubular intersticial.  Conclusión:  La detección temprana de dicho síndrome y el manejo oportuno tuvo gran impacto en la evolución clínica del paciente. Fue egresado a domicilio y recibió seguimiento por parte de los servicios de nefrología y hematología por consulta externa.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract: Fisher-Evans syndrome or Evans syndrome is the manifestation of immune hemolytic anemia that may be simultaneous or subsequent to immune thrombocytopenia. Because its diagnosis is not common, there are currently no exact information about its prevalence. According to its origin, it can be an idiopathic or secondary event. We present the case of a 58-year-old male, who developed Fisher-Evans syndrome associated to acute pancreatitis, in the intensive care unit (ICU); characterized by immune hemolytic anemia (Coombs positive) and thrombocytopenia; also presented renal dysfunction. During his hospital stay, he received steroid treatment; intravenous methylprednisolone for three days, oral prednisone (1 mg/kg/day) and human immunoglobulin (0.4 g/kg/day) for five days.  Results:  He presented clinical improvement, with resolution of anemia and thrombocytopenia. It was evaluated by the nephrology service; who established management with hemodialysis and performed renal biopsy with a report of interstitial tubular necrosis.  Conclusion:  Early detection of this syndrome and timely management had a great impact on the clinical evolution of the patient. He was graduated from home and received follow-up by the nephrology and hematology services by external.]]></p></abstract>
<abstract abstract-type="short" xml:lang="pt"><p><![CDATA[Resumo: A síndrome de Fisher-Evans ou Evans é a manifestação de anemia hemolítica imune que pode ser simultânea ou subsequente à trombocitopenia imune. Como seu diagnóstico não é comum, atualmente não há números exatos sobre sua prevalência. De acordo com sua origem, pode ser um evento idiopático ou secundário. Apresentamos o caso de um homem de 58 anos que desenvolveu a síndrome de Fisher-Evans associada a pancreatite aguda, internado em Unidade de Terapia Intensiva (UTI). Caracterizada por anemia hemolítica imunológica (Coombs positivo) e trombocitopenia; además o paciente apresentava disfunção renal. Durante sua internação, ele recebeu tratamento com esteróides; metilprednisolona intravenosa por 3 dias, prednisona oral (1 mg/kg/dia) e imunoglobulina humana (0.4 g/kg/dia) por 5 dias.  Resultados:  Apresentou melhora do quadro clínico, com resolução da anemia e trombocitopenia. Foi avaliado pelo serviço de nefrologia; que instituiu tratamento com hemodiálise e realizou biópsia renal com relato de necrose tubular intersticial.  Conclusão:  A detecção precoce desta síndrome e o manejo oportuno tiveram grande impacto na evolução clínica do paciente. Recebeu alta hospitalar e acompanhamento nos serviços de nefrologia e hematologia em consulta ambulatorial.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Síndrome de Fisher-Evans]]></kwd>
<kwd lng="es"><![CDATA[pancreatitis]]></kwd>
<kwd lng="es"><![CDATA[unidad de terapia intensiva]]></kwd>
<kwd lng="en"><![CDATA[Fisher-Evans syndrome]]></kwd>
<kwd lng="en"><![CDATA[pancreatitis]]></kwd>
<kwd lng="en"><![CDATA[intensive care unit]]></kwd>
<kwd lng="pt"><![CDATA[Síndrome de Fisher-Evans]]></kwd>
<kwd lng="pt"><![CDATA[pancreatite]]></kwd>
<kwd lng="pt"><![CDATA[Unidade de Terapia Intensiva]]></kwd>
</kwd-group>
</article-meta>
</front><back>
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