<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2007-4085</journal-id>
<journal-title><![CDATA[Revista mexicana de urología]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. mex. urol.]]></abbrev-journal-title>
<issn>2007-4085</issn>
<publisher>
<publisher-name><![CDATA[Sociedad Mexicana de Urología]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2007-40852021000200006</article-id>
<article-id pub-id-type="doi">10.48193/revistamexicanadeurologa.v81i2.678</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Paradigma actual de la cistopatía diabética en México]]></article-title>
<article-title xml:lang="en"><![CDATA[The current paradigm of diabetic cystopathy in Mexico]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Lira-Moreno]]></surname>
<given-names><![CDATA[Gonzalo]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Serrano]]></surname>
<given-names><![CDATA[Stefania]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Hernández-León]]></surname>
<given-names><![CDATA[Omar]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Castellanos]]></surname>
<given-names><![CDATA[Hibert]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Hospital Juárez de México  ]]></institution>
<addr-line><![CDATA[Ciudad de México ]]></addr-line>
<country>México</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>04</month>
<year>2021</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>04</month>
<year>2021</year>
</pub-date>
<volume>81</volume>
<numero>2</numero>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S2007-40852021000200006&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S2007-40852021000200006&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S2007-40852021000200006&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen El termino cistopatía diabética se atribuye al deterioro de la función del detrusor provocado por una combinación de mecanismos miogénicos y neurogénicos que dan como resultado el aumento de la capacidad y alteración en los volúmenes vesicales residuales postmiccionales, acompañado de una disminución en la sensación y contracción vesical en los pacientes diabéticos, siendo principalmente una enfermedad originada por la presencia de neuropatía de origen sensorial y autonómica por las alteraciones que sufren las fibras nerviosas vesicales. Clínicamente los pacientes cursan con una sintomatología insidiosa y cuando se presenta algún síntoma se encuentra en una fase avanzada, siendo la incontinencia urinaria el principal síntoma que se presenta. Su diagnóstico se establece mediante la presentación clínica asociada a sintomatología neurológica y se confirma con estudios de urodinamia. El control estricto y apego al tratamiento de la diabetes mellitus, es la piedra angular para evitar complicaciones a largo plazo como lo es la cistopatía diabética. Una vez que se establece la enfermedad hay diversas formas de tratamiento como puede ser farmacológico con una amplia variedad de fármacos a elegir y/o el manejo quirúrgico como la denervación vesical o la miomectomía.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract The term diabetic cystopathy is attributed to the deterioration of detrusor function caused by a combination of myogenic and neurogenic mechanisms, resulting in an increase in postmicturition residual bladder volume capacity and alteration, accompanied by reduced bladder contraction and sensation, in diabetic patients. The disease primarily originates from sensory and autonomic neuropathy due to alterations in the bladder nerve fibers. Clinical presentation is insidious, and when a symptom, usually urinary incontinence, does appear, the disease is in an advanced stage. Diagnosis is made through clinical presentation, associated with neurologic symptomatology, and confirmed through urodynamic studies. Strict control of diabetes mellitus, with treatment adherence, is essential for preventing complications in the long term, such as diabetic cystopathy. Once the disease is diagnosed, treatment can be pharmacologic, with a wide variety of drugs to choose from, and/or surgical, such as bladder denervation or myomectomy.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Cystopathy]]></kwd>
<kwd lng="en"><![CDATA[Urodynamics]]></kwd>
<kwd lng="en"><![CDATA[Voiding reflex]]></kwd>
<kwd lng="en"><![CDATA[Obstructive symptoms]]></kwd>
<kwd lng="en"><![CDATA[Urinary symptoms]]></kwd>
<kwd lng="en"><![CDATA[Cystopathy treatment]]></kwd>
<kwd lng="es"><![CDATA[Cistopatía diabética]]></kwd>
<kwd lng="es"><![CDATA[urodinamia]]></kwd>
<kwd lng="es"><![CDATA[reflejo miccional]]></kwd>
<kwd lng="es"><![CDATA[síntomas obstructivos]]></kwd>
<kwd lng="es"><![CDATA[síntomas urinarios]]></kwd>
<kwd lng="es"><![CDATA[tratamiento cistopatía]]></kwd>
</kwd-group>
</article-meta>
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