<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1405-888X</journal-id>
<journal-title><![CDATA[TIP. Revista especializada en ciencias químico-biológicas]]></journal-title>
<abbrev-journal-title><![CDATA[TIP]]></abbrev-journal-title>
<issn>1405-888X</issn>
<publisher>
<publisher-name><![CDATA[Universidad Nacional Autónoma de México, Facultad de Estudios Superiores Zaragoza]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1405-888X2023000100202</article-id>
<article-id pub-id-type="doi">10.22201/fesz.23958723e.2023.547</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Las mitocondrias: sus funciones, las relaciones con otros organelos, la supervivencia celular y la medicina mitocondrial]]></article-title>
<article-title xml:lang="en"><![CDATA[Mitochondrion: their functions, relationships with cellular organelles, cell survival and mitochondrial medicine]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Olvera-Sánchez]]></surname>
<given-names><![CDATA[Sofía]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Gómez-Chang]]></surname>
<given-names><![CDATA[Erika]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Flores-Herrera]]></surname>
<given-names><![CDATA[Oscar]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Martínez]]></surname>
<given-names><![CDATA[Federico]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Nacional Autónoma de México Facultad de Medicina Departamento de Bioquímica]]></institution>
<addr-line><![CDATA[Coyoacán Ciudad de México]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>00</month>
<year>2023</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>00</month>
<year>2023</year>
</pub-date>
<volume>26</volume>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S1405-888X2023000100202&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S1405-888X2023000100202&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S1405-888X2023000100202&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen Durante años se consideró que la función más importante de las mitocondrias era la de proveer a la célula de energía en forma de ATP o incluso, fungir como amortiguadores de la concentración del calcio citosólico. Sin embargo, se ha demostrado que las mitocondrias tienen más atributos al ser una parte medular de las cascadas de señalización, y de compartir funciones con las estructuras intra y extracelulares que permiten mantener la homeostasis y la función celular. Además, hay reportes que muestran que las células sanas pueden transferir a las mitocondrias por el torrente circulatorio o directamente a las células dañadas con el objetivo de brindarles protección y mejorar su función bioenergética en condiciones específicas. Los mecanismos de señalización para el reconocimiento de moléculas marcadoras son determinantes para establecer la transferencia de las mitocondrias entre células y a través de las estructuras vesiculares o de los túneles (TNT), entre otros sistemas. Actualmente, se han desarrollado estrategias para el aislamiento de las mitocondrias, las cuales, a su vez, se inyectan en los tejidos permitiéndoles su recuperación funcional, a lo que se ha llamado la &#8220;medicina mitocondrial&#8221; o la &#8220;mitocuración&#8221;. Aunque estos hallazgos sugieren que las mitocondrias podrían ser un nuevo método de profilaxis para las enfermedades o trastornos metabólicos, es necesario considerar que la transferencia mitocondrial no es del todo una solución positiva en el tratamiento de algunos padecimientos.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract For years, it was considered that the most important function of mitochondria was to provide the cell with energy in the form of ATP or even to function as a buffer of the cytosolic calcium concentration. However, it has been shown that mitochondria are a key part of signaling cascades or either, they establish shared functions with intra- and extracellular structures, allowing to maintain homeostasis and cell function. In addition, there are several reports showing that healthy cells are capable of transferring mitochondria to the bloodstream or to damaged cells with the aim of providing protection or to improve their bioenergetic condition under specific conditions. The signaling mechanisms for the recognition of marker molecules are decisive to establish the transfer of mitochondria between cells through vesicular structures or tunnels (TNT), among other systems. Currently, it has been developed strategies for mitochondrial isolation, which in turn, are injected into the tissues allowing their functional recovery, opening the door to what has been called &#8220;mitochondrial medicine&#8221; or &#8220;mitocure&#8221;. Although these findings suggest that mitochondria could be a new prophylactic method for diseases or metabolic disorders, it is also necessary to analyze mitochondrial transfer may not always be positive for the organism in certain ailments.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Mitocondrias]]></kwd>
<kwd lng="es"><![CDATA[estructura mitocondrial]]></kwd>
<kwd lng="es"><![CDATA[transferencia de mitocondrias]]></kwd>
<kwd lng="es"><![CDATA[liberación celular de mitocondrias]]></kwd>
<kwd lng="es"><![CDATA[medicina mitocondrial]]></kwd>
<kwd lng="es"><![CDATA[regeneración funcional de las células]]></kwd>
<kwd lng="en"><![CDATA[Mitochondria]]></kwd>
<kwd lng="en"><![CDATA[mitochondrial structure]]></kwd>
<kwd lng="en"><![CDATA[mitochondrial transfer]]></kwd>
<kwd lng="en"><![CDATA[mitochondrial cell release]]></kwd>
<kwd lng="en"><![CDATA[mitochondrial medicine]]></kwd>
<kwd lng="en"><![CDATA[functional cell regeneration]]></kwd>
</kwd-group>
</article-meta>
</front><back>
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