<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0186-4866</journal-id>
<journal-title><![CDATA[Medicina interna de México]]></journal-title>
<abbrev-journal-title><![CDATA[Med. interna Méx.]]></abbrev-journal-title>
<issn>0186-4866</issn>
<publisher>
<publisher-name><![CDATA[Edición y Farmacia S.A. de C.V.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0186-48662017000500563</article-id>
<article-id pub-id-type="doi">10.24245/mim.v33i5.1042</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Deshidrogenasa láctica como factor pronóstico en neumonías]]></article-title>
<article-title xml:lang="en"><![CDATA[Lactic dehydrogenase as a prognostic factor in pneumonias]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[García-Arroyo]]></surname>
<given-names><![CDATA[A.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Rosas-Dossetti]]></surname>
<given-names><![CDATA[M.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Santoyo-Sánchez]]></surname>
<given-names><![CDATA[A.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Salcedo-Roldán]]></surname>
<given-names><![CDATA[M.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Ramos-Peñafiel]]></surname>
<given-names><![CDATA[CO.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Instituto de Salud del Estado de México Hospital General de Cuautitlán Servicio de Medicina Interna]]></institution>
<addr-line><![CDATA[ Estado de México]]></addr-line>
</aff>
<aff id="Af2">
<institution><![CDATA[,Instituto de Salud del Estado de México Hospital General de Cuautitlán Departamento de Epidemiología]]></institution>
<addr-line><![CDATA[ Estado de México]]></addr-line>
</aff>
<aff id="Af3">
<institution><![CDATA[,Hospital General de México Dr. Eduardo Liceaga  ]]></institution>
<addr-line><![CDATA[Ciudad de México ]]></addr-line>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>10</month>
<year>2017</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>10</month>
<year>2017</year>
</pub-date>
<volume>33</volume>
<numero>5</numero>
<fpage>563</fpage>
<lpage>571</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_arttext&amp;pid=S0186-48662017000500563&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_abstract&amp;pid=S0186-48662017000500563&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.mx/scielo.php?script=sci_pdf&amp;pid=S0186-48662017000500563&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen  ANTECEDENTES: la pandemia de influenza en 2009 renovó el interés por identificar oportunamente casos sospechosos de influenza mediante estudios de laboratorio rutinarios, uno de los más estudiados es la deshidrogenasa láctica (DHL).  OBJETIVO: determinar si los pacientes con neumonía por influenza A (H1N1) tienen alteraciones particulares en estudios rutinarios de laboratorio, particularmente en concentraciones de DHL y analizar la implicación pronóstica.  MATERIAL Y MÉTODO: estudio de casos y controles de pacientes con diagnóstico confirmado de neumonía por influenza A (H1N1) [caso], y pacientes con neumonía bacteriana (control) atendidos de diciembre de 2013 a julio de 2014.  RESULTADOS: se analizaron 31 casos, 45% (n = 14) tenían diagnóstico de neumonía por el virus de la influenza A (H1N1), el 55% restante (n = 17) se consideró de causa bacteriana. La media de edad fue de 38 años (límites: 16-62). Las concentraciones de DHL al momento del diagnóstico fueron, en promedio, de 578.77 UI/L (límites: 191-1096), fue mayor en el grupo con neumonía por influenza A (H1N1) [573 vs 624.7 UI/L, p = 0.366]. En el análisis global las concentraciones de DHL &gt; 350 UI/L al diagnóstico y fin del tratamiento repercutieron fuertemente de manera negativa en la mortalidad (OR: 84.0, IC95%: 4.4754-1576.6044 y OR: 154.0, 8.6261-2749.3255). La supervivencia general fue de 18 días, menor en el grupo de A (H1N1) [4 vs 25 días, p = 0.016).  CONCLUSIONES: las concentraciones de DHL &gt; 350 UI/L pueden considerarse un biomarcador de gravedad y repercuten negativamente en la supervivencia de pacientes con neumonía, sin poder discriminar al posible agente etiológico.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract  BACKGROUND: The 2009 influenza pandemic renewed interest in timely identification of suspected influenza cases through routine laboratory studies, the most studied is lactic dehydrogenase (DHL).  OBJECTIVE: To determine if patients with influenza A (H1N1) pneumonia present particular alterations inside routine laboratory studies, particularly in DHL levels and analyze the prognostic implication.  MATERIAL AND METHOD: A case-control study of patients with confirmed diagnosis of influenza A (H1N1) pneumonia (case), and patients with bacterial pneumonia (control) treated from December 2013 to July 2014.  RESULTS: Thirty-one cases were analyzed, 45% (n = 14) had a diagnosis of influenza A (H1N1) pneumonia, the remaining 55% (n = 17) was considered of bacterial etiology. The mean age was 38 (16-62) years old. The DHL level at diagnosis time was on average 578.77 IU/L (191-1096), higher in the group with influenza A (H1N1) pneumonia (573 IU/L vs 624.7 IU/L, p = 0.366). In the overall analysis, the levels of DHL &gt; 350 IU/L at diagnosis time and at the end of treatment had a negative impact on mortality (OR: 84.0, 95%CI: 4.4754-1576.6044, and OR: 154.0, 8.6261-2749.3255). Overall survival was 18 days, lower in the A (H1N1) group (4 vs 25 days, p = 0.016).  CONCLUSIONS: DHL &gt; 350 IU/L can be considered a severity biomarker, also has a negative impact on the survival of patients with pneumonia without being able to discriminate the possible etiological agent.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[L-lactato deshidrogenasa]]></kwd>
<kwd lng="es"><![CDATA[análisis de supervivencia]]></kwd>
<kwd lng="es"><![CDATA[subtipo H1N1 del virus de la influenza A]]></kwd>
<kwd lng="es"><![CDATA[neumonía bacteriana]]></kwd>
<kwd lng="es"><![CDATA[neumonía]]></kwd>
<kwd lng="es"><![CDATA[pronóstico]]></kwd>
<kwd lng="en"><![CDATA[L-lactate dehydrogenase]]></kwd>
<kwd lng="en"><![CDATA[survival analysis]]></kwd>
<kwd lng="en"><![CDATA[influenza A virus]]></kwd>
<kwd lng="en"><![CDATA[H1N1 subtype]]></kwd>
<kwd lng="en"><![CDATA[pneumonia]]></kwd>
<kwd lng="en"><![CDATA[bacterial]]></kwd>
<kwd lng="en"><![CDATA[pneumonia]]></kwd>
<kwd lng="en"><![CDATA[prognosis]]></kwd>
</kwd-group>
</article-meta>
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